BCL2 is a downstream effector of MIZ-1 essential for blocking c-MYC-induced apoptosis.
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Miz1 and HectH9 regulate the stability of the checkpoint protein, TopBP1The Bcl-2 apoptotic switch in cancer development and therapyGenetic and Epigenetic Mechanisms That Maintain Hematopoietic Stem Cell FunctionThe role of MIZ-1 in MYC-dependent tumorigenesisMiz1 is a critical repressor of cdkn1a during skin tumorigenesisMyc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addictionNac1 interacts with the POZ-domain transcription factor, Miz1.MYC association with cancer risk and a new model of MYC-mediated repression.MYC cofactors: molecular switches controlling diverse biological outcomes.A tumor suppressor role for PP2A-B56alpha through negative regulation of c-Myc and other key oncoproteins.Regulation of hedgehog signaling by Myc-interacting zinc finger protein 1, Miz1ARF antagonizes the ability of Miz-1 to inhibit p53-mediated transactivation.Molecular mechanisms of cigarette smoke-induced proliferation of lung cells and prevention by vitamin C.Inhibition of the single downstream target BAG1 activates the latent apoptotic potential of MYC.Repression of SRF target genes is critical for Myc-dependent apoptosis of epithelial cells.Prognostic impact of concurrent MYC and BCL6 rearrangements and expression in de novo diffuse large B-cell lymphoma.BCL6 suppression of BCL2 via Miz1 and its disruption in diffuse large B cell lymphoma.Apoptotic signaling by c-MYC.Genome-wide identification of post-translational modulators of transcription factor activity in human B cells.Regulation of CDKN2B expression by interaction of Arnt with Miz-1--a basis for functional integration between the HIF and Myc gene regulatory pathways.Myc Induces miRNA-Mediated Apoptosis in Response to HDAC Inhibition in Hematologic Malignancies.Dock3 attenuates neural cell death due to NMDA neurotoxicity and oxidative stress in a mouse model of normal tension glaucoma.Transcriptional regulation of Wnt inhibitory factor-1 by Miz-1/c-Myc.The Arf tumor suppressor protein inhibits Miz1 to suppress cell adhesion and induce apoptosis.Cytosolic acetyl-CoA synthetase affected tumor cell survival under hypoxia: the possible function in tumor acetyl-CoA/acetate metabolism.An N-Myc truncation analogous to c-Myc-S induces cell proliferation independently of transactivation but dependent on Myc homology box II.Nuclear factor-kappa B1 inhibits early apoptosis of glioma cells by promoting the expression of Bcl-2Emerging Concepts in the Analysis of Transcriptional Targets of the MYC Oncoprotein: Are the Targets Targetable?Decreased MIZ1 Expression in Severe Experimental Acute Pancreatitis: A Rat Study.
P2860
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P2860
BCL2 is a downstream effector of MIZ-1 essential for blocking c-MYC-induced apoptosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
BCL2 is a downstream effector of MIZ-1 essential for blocking c-MYC-induced apoptosis.
@en
type
label
BCL2 is a downstream effector of MIZ-1 essential for blocking c-MYC-induced apoptosis.
@en
prefLabel
BCL2 is a downstream effector of MIZ-1 essential for blocking c-MYC-induced apoptosis.
@en
P2860
P356
P1476
BCL2 is a downstream effector of MIZ-1 essential for blocking c-MYC-induced apoptosis.
@en
P2093
Jagruti H Patel
Steven B McMahon
P2860
P356
10.1074/JBC.M609138200
P407
P577
2006-11-01T00:00:00Z