Secreted frizzled-related protein 4 is a negative regulator of peak BMD in SAMP6 mice.
about
Lrp5 functions in bone to regulate bone massCthrc1 is a positive regulator of osteoblastic bone formationMouse genome-wide association and systems genetics identify Asxl2 as a regulator of bone mineral density and osteoclastogenesisTenascin-W inhibits proliferation and differentiation of preosteoblasts during endochondral bone formationExome sequencing of senescence-accelerated mice (SAM) reveals deleterious mutations in degenerative disease-causing genes.Impact of the environment on the skeleton: is it modulated by genetic factors?Characterization of age-related gene expression profiling in bone marrow and epididymal adipocytes.Quantitative trait loci, genes, and polymorphisms that regulate bone mineral density in mouseMaf promotes osteoblast differentiation in mice by mediating the age-related switch in mesenchymal cell differentiation.Aging and bone.Screening of differentially expressed genes in the growth plate of broiler chickens with tibial dyschondroplasia by microarray analysis.Fibroblast growth factor 2 stimulation of osteoblast differentiation and bone formation is mediated by modulation of the Wnt signaling pathway.TGF-β regulates sclerostin expression via the ECR5 enhancer.A Comprehensive Overview of Skeletal Phenotypes Associated with Alterations in Wnt/β-catenin Signaling in Humans and Mice.Role of fibroblast growth factor 2 and Wnt signaling in anabolic effects of parathyroid hormone on bone formation.Update on Wnt signaling in bone cell biology and bone disease.Building bone to reverse osteoporosis and repair fracturessFRP4-dependent Wnt signal modulation is critical for bone remodeling during postnatal development and age-related bone lossAge-dependent Wnt gene expression in bone and during the course of osteoblast differentiation.The need for mouse models in osteoporosis genetics research.The relevance of mouse models for investigating age-related bone loss in humans.Resorption controls bone anabolism driven by parathyroid hormone (PTH) receptor signaling in osteocytesMechanical stimulation of bone formation is normal in the SAMP6 mouse.How genomics has informed our understanding of the pathogenesis of osteoporosis.Targeting the Wnt signaling pathway to augment bone formation.Treadmill running exercise prevents senile osteoporosis and upregulates the Wnt signaling pathway in SAMP6 mice.Insights into the genetics of osteoporosis from recent genome-wide association studies.Characterization of senescence-accelerated mouse prone 6 (SAMP6) as an animal model for brain research.Osteoporotic Animal Models of Bone Healing: Advantages and Pitfalls.Human epicardial adipose tissue-derived and circulating secreted frizzled-related protein 4 (SFRP4) levels are increased in patients with coronary artery disease.Bone mineral density is linked to 1p36 and 7p15-13 in a southern Chinese population.
P2860
Q24615653-C8F8AA96-0002-4A6B-B649-0FCD64C1718FQ28511903-EDB04CA1-ADCE-49D1-AA41-8AE177FC243FQ28593424-A723D1D9-0F8D-46D7-AC84-BEAAB610C94BQ28854386-9A1804A9-5024-4ACA-BF8E-C680CB9E858DQ30455193-AAC5251B-EAC9-432E-90C2-A4D99F2F6F89Q33684679-28FF79AC-4DA2-4FAE-B8E3-93E07374D8C1Q33890592-CC69D606-42B8-4D15-BB88-ABE9400E36DBQ33980500-37C8D052-73B3-4C4D-829D-D19977A6BFC1Q34162157-7192B2C3-3138-4440-B935-5CC0C901DBE9Q34348145-BED45352-9425-4035-8819-14B5D36C412DQ34687218-703C82CA-196E-427D-A524-020198A1FFA7Q35562548-0E420853-5914-470C-A0DD-C6C7511CFA3FQ35754103-ACE257FB-B078-4BFF-A51F-D49E3D496DB6Q35755034-2D807414-3FED-4F90-AEF7-13AD03E7EC95Q36020434-34F38602-E2B8-4D67-AF9F-4F47AF143E41Q36083303-D4D98944-5072-4FD5-BCED-F1A760A84060Q36407024-38F80402-305C-4966-83A0-A7BBC72BB9B7Q36841079-B9DC6CFB-7058-4465-979E-1C6E260C1A75Q36979410-A7808519-A7D8-4891-AE25-031A1701DD85Q37056296-3ED1FFF8-126D-4180-9666-B5E3C73EDA54Q37189204-67A6A8FA-FA6B-4D62-A8CC-0C6A6BA871F1Q37226223-F4D1D75E-DDE4-455D-AB32-28D5F24F5E4CQ37251092-B63018EA-8B46-4115-8D03-0B2F88E64234Q37402642-79613BC0-3455-43D6-8C9B-C269DC73E8E5Q37479050-D903C710-1EC6-44AC-A9DD-755990EDA169Q37687575-AC2DD016-B94A-4A6D-B895-E5069119A25AQ37922222-1B888D0A-0419-4EE8-A521-6213857F93FEQ38187530-0B032C40-81E4-43D7-BAEA-8CFA07A38749Q38785762-120E1ECD-2B5D-40E5-8EE4-9F93D6097034Q42379235-016095A0-5F49-4C0F-A2AA-A87254ACDAA0Q51554730-54EC92D4-1D6B-4BCF-B380-2DA9C10445BA
P2860
Secreted frizzled-related protein 4 is a negative regulator of peak BMD in SAMP6 mice.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Secreted frizzled-related protein 4 is a negative regulator of peak BMD in SAMP6 mice.
@en
type
label
Secreted frizzled-related protein 4 is a negative regulator of peak BMD in SAMP6 mice.
@en
prefLabel
Secreted frizzled-related protein 4 is a negative regulator of peak BMD in SAMP6 mice.
@en
P2093
P356
P1476
Secreted frizzled-related protein 4 is a negative regulator of peak BMD in SAMP6 mice
@en
P2093
Haruhiko Akiyama
Keiichi Higuchi
Maiko Hashimoto
Masanori Hosokawa
Masayuki Mori
Motoyuki Shimizu
Rika Nakanishi
Shuzo Okudaira
Tadao Tsuboyama
Takashi Nakamura
P304
P356
10.1359/JBMR.060719
P50
P577
2006-11-01T00:00:00Z