Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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Resveratrol modulates GSH system in C6 astroglial cells through heme oxygenase 1 pathway.Recent advances in hepatic encephalopathy.

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Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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P1476

Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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P2093

Carlos-Alberto Gonçalves

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Diogo Onofre Souza

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Larissa Daniele Bobermin

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Marina Concli Leite

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P2860

Tripartite synapses: astrocytes process and control synaptic information

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Brain energy metabolism and mitochondrial dysfunction in acute and chronic hepatic encephalopathy

Glial cells in (patho)physiology

Astrocytes: biology and pathology

Astrocytes are active players in cerebral innate immunity

Neuroinflammation leads to region-dependent alterations in astrocyte gap junction communication and hemichannel activity.

Pathophysiology of brain dysfunction in hyperammonemic syndromes: The many faces of glutamine.

Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice.

Gene expression profiling of astrocytes from hyperammonemic mice reveals altered pathways for water and potassium homeostasis in vivo.

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s12640-015-9581-5

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314-324

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scholarly article

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10.1007/S12640-015-9581-5

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P478

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Bernardo A Arús

André Quincozes-Santos

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2015-12-08T00:00:00Z

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26646155

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P921

neurotoxicity

Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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P2860

P2860

Gap Junction Intercellular Communication Mediates Ammonia-Induced Neurotoxicity.

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