Endogenous expression of phosphorylated histone H2AX in tumors in relation to DNA double-strand breaks and genomic instability.
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Proteomic dissection of cell type-specific H2AX-interacting protein complex associated with hepatocellular carcinomaCurrent Management Strategies in Breast Cancer by Targeting Key Altered Molecular PlayersExpression and functionality of histone H2A variants in cancerComputational analysis of the number, area and density of gamma-H2AX foci in breast cancer cells exposed to (111)In-DTPA-hEGF or gamma-rays using Image-J softwareGammaH2AX and cancerMicrowaves from Mobile Phones Inhibit 53BP1 Focus Formation in Human Stem Cells More Strongly Than in Differentiated Cells: Possible Mechanistic Link to Cancer RiskKinetics of the UV-induced DNA damage response in relation to cell cycle phase. Correlation with DNA replication.Molecular markers of radiation-related normal tissue toxicity.Telomere-dependent and telomere-independent origins of endogenous DNA damage in tumor cells.pATM and γH2AX are effective radiation biomarkers in assessing the radiosensitivity of 12C6+ in human tumor cells.Centromere-localized breaks indicate the generation of DNA damage by the mitotic spindle.Combined PTEN Mutation and Protein Expression Associate with Overall and Disease-Free Survival of Glioblastoma Patients.Role of oxidatively induced DNA lesions in human pathogenesisHistone gammaH2AX and poly(ADP-ribose) as clinical pharmacodynamic biomarkers.Opposite roles for p38MAPK-driven responses and reactive oxygen species in the persistence and resolution of radiation-induced genomic instability.Evaluation of the efficacy of radiation-modifying compounds using γH2AX as a molecular marker of DNA double-strand breaksHuman APOBEC3A isoforms translocate to the nucleus and induce DNA double strand breaks leading to cell stress and death.H2AX: functional roles and potential applications.Dysregulation of PAK1 Is Associated with DNA Damage and Is of Prognostic Importance in Primary Esophageal Small Cell Carcinoma.Heterogeneity of Genetic Damage in Cervical Nuclei and Lymphocytes in Women with Different Levels of Dysplasia and Cancer-Associated Risk Factors.H2AX phosphorylation at the sites of DNA double-strand breaks in cultivated mammalian cells and tissues.Intrinsic Radiosensitivity and Cellular Characterization of 27 Canine Cancer Cell Lines.Chromatin structure and DNA double-strand break responses in cancer progression and therapy.Expression of γH2AX may help in defining a genetically more stable subtype of infiltrating ductal carcinoma of breast.The role of BRAF mutation and p53 inactivation during transformation of a subpopulation of primary human melanocytesThe contribution of women to radiobiology: Marie Curie and beyondRisk assessment of esophageal adenocarcinoma using γ-H2AX assayEvidence for ongoing DNA damage in multiple myeloma cells as revealed by constitutive phosphorylation of H2AXSpatiotemporal characterization of ionizing radiation induced DNA damage foci and their relation to chromatin organizationH2AX a promising biomarker for lung cancer: a review.TPX2: of spindle assembly, DNA damage response, and cancer.Complex DNA Damage: A Route to Radiation-Induced Genomic Instability and Carcinogenesis.Spontaneous γH2AX Foci in Human Solid Tumor-Derived Cell Lines in Relation to p21WAF1 and WIP1 Expression.Does the number of irradiated cells influence the spatial distribution of bystander effects?A computational platform for robotized fluorescence microscopy (II): DNA damage, replication, checkpoint activation, and cell cycle progression by high-content high-resolution multiparameter image-cytometry.Downregulation of XPF-ERCC1 enhances cisplatin efficacy in cancer cells.DNA repair foci and late apoptosis/necrosis in peripheral blood lymphocytes of breast cancer patients undergoing radiotherapy.Genome Instability and γH2AX.Differences in Phosphorylated Histone H2AX Foci Formation and Removal of Cells Exposed to Low and High Linear Energy Transfer Radiation.Endogenous DNA breaks: gammaH2AX and the role of telomeres.
P2860
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P2860
Endogenous expression of phosphorylated histone H2AX in tumors in relation to DNA double-strand breaks and genomic instability.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Endogenous expression of phosp ...... reaks and genomic instability.
@en
type
label
Endogenous expression of phosp ...... reaks and genomic instability.
@en
prefLabel
Endogenous expression of phosp ...... reaks and genomic instability.
@en
P2093
P1433
P1476
Endogenous expression of phosp ...... breaks and genomic instability
@en
P2093
P304
P356
10.1016/J.DNAREP.2006.05.040
P577
2006-06-30T00:00:00Z