An assessment of the role of reactive oxygen species and redox signaling in norepinephrine-induced apoptosis and hypertrophy of H9c2 cardiac myoblasts.
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Toll-like receptor 4 inhibition within the paraventricular nucleus attenuates blood pressure and inflammatory response in a genetic model of hypertensionCatecholamine stress hormones regulate cellular iron homeostasis by a posttranscriptional mechanism mediated by iron regulatory protein: implication in energy homeostasis.Norepinephrine-induced apoptotic and hypertrophic responses in H9c2 cardiac myoblasts are characterized by different repertoire of reactive oxygen species generationIschemia-reperfusion and cardioprotection: a delicate balance between reactive oxygen species generation and redox homeostasis.Heme oxygenase-1 inhibits pro-oxidant induced hypertrophy in HL-1 cardiomyocytes.Oxidative stress and β-amyloid protein in Alzheimer's disease.Regulation of β-adrenergic receptor function: an emphasis on receptor resensitization.Oxidative stress and cardiac hypertrophy: a review.Dopamine induces lipid accumulation, NADPH oxidase-related oxidative stress, and a proinflammatory status of the plasma membrane in H9c2 cells.Apelin-13 promotes cardiomyocyte hypertrophy via PI3K-Akt-ERK1/2-p70S6K and PI3K-induced autophagy.Central blockade of TLR4 improves cardiac function and attenuates myocardial inflammation in angiotensin II-induced hypertension.Cardiomyocyte H9c2 Cells Exhibit Differential Sensitivity to Intracellular Reactive Oxygen Species Generation with Regard to Their Hypertrophic vs Death Responses to Exogenously Added Hydrogen Peroxide.Antifungal activity of α-methyl trans cinnamaldehyde, its ligand and metal complexes: promising growth and ergosterol inhibitors.Crocetin attenuates norepinephrine-induced cytotoxicity in primary cultured rat cardiac myocytes by antioxidant in vitro.Activation of adrenergic receptor in H9c2 cardiac myoblasts co-stimulates Nox2 and the derived ROS mediate the downstream responses.Preclinical cardiac involvement in phaeochromocytoma: a study with integrated backscatter.Naphthoquinoxaline metabolite of mitoxantrone is less cardiotoxic than the parent compound and it can be a more cardiosafe drug in anticancer therapy.Relaxin ameliorates high glucose-induced cardiomyocyte hypertrophy and apoptosis via the Notch1 pathway.Decreased Nox4 levels in the myocardium of patients with aortic valve stenosis.A pro-survival effect of polyamine depletion on norepinephrine-mediated apoptosis in cardiac cells: role of signaling enzymes.
P2860
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P2860
An assessment of the role of reactive oxygen species and redox signaling in norepinephrine-induced apoptosis and hypertrophy of H9c2 cardiac myoblasts.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
An assessment of the role of r ...... phy of H9c2 cardiac myoblasts.
@en
type
label
An assessment of the role of r ...... phy of H9c2 cardiac myoblasts.
@en
prefLabel
An assessment of the role of r ...... phy of H9c2 cardiac myoblasts.
@en
P2093
P356
P1476
An assessment of the role of r ...... phy of H9c2 cardiac myoblasts.
@en
P2093
Amit Dinda
Chinmay K Mukhopadhyay
Manveen K Gupta
Mishra Sanghamitra
Rakesh K Tyagi
Shyamal K Goswami
Subir Maulik
T V Neelakantan
P304
P356
10.1089/ARS.2006.8.1081
P577
2006-05-01T00:00:00Z