Studies on avian infectious bronchitis virus (IBV). III. Interferon induction by and sensitivity to interferon of IBV.
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The zinc finger antiviral protein acts synergistically with an interferon-induced factor for maximal activity against alphavirusesA Single Amino Acid Substitution in the West Nile Virus Nonstructural Protein NS2A Disables Its Ability To Inhibit Alpha/Beta Interferon Induction and Attenuates Virus Virulence in MiceA mouse cell-adapted NS4B mutation attenuates West Nile virus RNA synthesisADP-ribosylhydrolase activity of Chikungunya virus macrodomain is critical for virus replication and virulenceMultiple interferon stimulated genes synergize with the zinc finger antiviral protein to mediate anti-alphavirus activity.Generation of mutant Uukuniemi viruses lacking the nonstructural protein NSs by reverse genetics indicates that NSs is a weak interferon antagonist.Inhibition of dengue virus by targeting viral NS4B proteinIn vitro and in ovo expression of chicken gamma interferon by a defective RNA of avian coronavirus infectious bronchitis virus.Mutation of Putative N-Glycosylation Sites on Dengue Virus NS4B Decreases RNA ReplicationHigh-Throughput Single-Cell Kinetics of Virus Infections in the Presence of Defective Interfering Particles.The Interferon Type I/III Response to Respiratory Syncytial Virus Infection in Airway Epithelial Cells Can Be Attenuated or Amplified by Antiviral TreatmentIncreased early RNA replication by chimeric West Nile virus W956IC leads to IPS-1-mediated activation of NF-κB and insufficient virus-mediated counteraction of the resulting canonical type I interferon signalingAnalysis of adaptive mutations in Kunjin virus replicon RNA reveals a novel role for the flavivirus nonstructural protein NS2A in inhibition of beta interferon promoter-driven transcription.Establishment of persistent foot-and-mouth disease virus (FMDV) infection in MDBK cells.Inhibition of infection spread by co-transmitted defective interfering particles.Infectious bronchitis virus: Immunopathogenesis of infection in the chicken.Infectious Bronchitis Coronavirus Inhibits STAT1 Signaling and Requires Accessory Proteins for Resistance to Type I Interferon Activity.Avian infectious bronchitis virus disrupts the melanoma differentiation associated gene 5 (MDA5) signaling pathway by cleavage of the adaptor protein MAVS.Use of reverse transcriptase-polymerase chain reaction-restriction fragment length polymorphism to examine the interaction between infectious bronchitis virus strains Massachusetts 41 and JMK in ovo.Leukocyte subpopulations in kidney and trachea of chickens infected with infectious bronchitis virus.Constitutively elevated levels of SOCS1 suppress innate responses in DF-1 immortalised chicken fibroblast cells.
P2860
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P2860
Studies on avian infectious bronchitis virus (IBV). III. Interferon induction by and sensitivity to interferon of IBV.
description
1979 nî lūn-bûn
@nan
1979年の論文
@ja
1979年論文
@yue
1979年論文
@zh-hant
1979年論文
@zh-hk
1979年論文
@zh-mo
1979年論文
@zh-tw
1979年论文
@wuu
1979年论文
@zh
1979年论文
@zh-cn
name
Studies on avian infectious br ...... sitivity to interferon of IBV.
@en
type
label
Studies on avian infectious br ...... sitivity to interferon of IBV.
@en
prefLabel
Studies on avian infectious br ...... sitivity to interferon of IBV.
@en
P2093
P356
P1433
P1476
Studies on avian infectious br ...... sitivity to interferon of IBV.
@en
P2093
P2888
P304
P356
10.1007/BF01317496
P577
1979-01-01T00:00:00Z
P6179
1009567287