Upregulation of persistent and ramp sodium current in dorsal horn neurons after spinal cord injury.
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Mechanisms of chronic central neuropathic pain after spinal cord injuryChanging channels in pain and epilepsy: Exploiting ion channel gene therapy for disorders of neuronal hyperexcitabilitySynergetic action of domain II and IV underlies persistent current generation in Nav1.3 as revealed by a tarantula toxin.Acute cocaine exposure elicits rises in calcium in arousal-related laterodorsal tegmental neuronsBlockade of persistent sodium currents contributes to the riluzole-induced inhibition of spontaneous activity and oscillations in injured DRG neurons.Regulatory effects of intermittent noxious stimulation on spinal cord injury-sensitive microRNAs and their presumptive targets following spinal cord contusionTetrodotoxin (TTX) as a therapeutic agent for pain.Pretreatment with intrathecal amitriptyline potentiates anti-hyperalgesic effects of post-injury intra-peritoneal amitriptyline following spinal nerve ligationRemote astrocytic and microglial activation modulates neuronal hyperexcitability and below-level neuropathic pain after spinal injury in ratCalcium channel alpha-2-delta-1 protein upregulation in dorsal spinal cord mediates spinal cord injury-induced neuropathic pain states.The roles of sodium channels in nociception: Implications for mechanisms of painSodium channel expression in the ventral posterolateral nucleus of the thalamus after peripheral nerve injuryIntermittent noxious stimulation following spinal cord contusion injury impairs locomotor recovery and reduces spinal brain-derived neurotrophic factor-tropomyosin-receptor kinase signaling in adult rats.Sequence variations at I260 and A1731 contribute to persistent currents in Drosophila sodium channels.Conotoxins targeting neuronal voltage-gated sodium channel subtypes: potential analgesics?Virus-mediated shRNA knockdown of Na(v)1.3 in rat dorsal root ganglion attenuates nerve injury-induced neuropathic painTranscriptional expression of voltage-gated Na⁺ and voltage-independent K⁺ channels in the developing rat superficial dorsal horn.Multiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons.Blocking sodium channels to treat neuropathic pain.Mutant SOD1 protein increases Nav1.3 channel excitabilityKnockout of Cyclophilin-D Provides Partial Amelioration of Intrinsic and Synaptic Properties Altered by Mild Traumatic Brain Injury.Novel SCN3A variants associated with focal epilepsy in children.Ion channel blockers for the treatment of neuropathic pain.Recent developments regarding voltage-gated sodium channel blockers for the treatment of inherited and acquired neuropathic pain syndromes.Sodium channels, the electrogenisome and the electrogenistat: lessons and questions from the clinic.Advances in targeting voltage-gated sodium channels with small molecules.Opposite, bidirectional shifts in excitation and inhibition in specific types of dorsal horn interneurons are associated with spasticity and pain post-SCI.Cooperativity between remote sites of ectopic spiking allows afterdischarge to be initiated and maintained at different locationsPharmacological kinetics of BmK AS, a sodium channel site 4-specific modulator on Nav1.3.Neuronal-Glial Interactions Maintain Chronic Neuropathic Pain after Spinal Cord Injury.Below level central pain induced by discrete dorsal spinal cord injuryUnilateral T13 and L1 dorsal root avulsion: methods for a novel model of central neuropathic pain.Changes of voltage-gated sodium channels in sensory nerve regeneration and neuropathic pain models.Distribution and function of voltage-gated sodium channels in the nervous system.Cleavage of Na(+) channels by calpain increases persistent Na(+) current and promotes spasticity after spinal cord injury.Reduced excitability of gp130-deficient nociceptors is associated with increased voltage-gated potassium currents and Kcna4 channel upregulation.Mutations in SCN3A cause early infantile epileptic encephalopathyIonic Homeostasis Maintenance in ALS: Focus on New Therapeutic TargetsAddressing the Issue of Tetrodotoxin Targeting
P2860
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P2860
Upregulation of persistent and ramp sodium current in dorsal horn neurons after spinal cord injury.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Upregulation of persistent and ...... rons after spinal cord injury.
@en
type
label
Upregulation of persistent and ...... rons after spinal cord injury.
@en
prefLabel
Upregulation of persistent and ...... rons after spinal cord injury.
@en
P1476
Upregulation of persistent and ...... urons after spinal cord injury
@en
P2093
Bryan C Hains
Stephen G Waxman
P2888
P304
P356
10.1007/S00221-006-0511-X
P577
2006-05-23T00:00:00Z