An enzyme-linked immunosorbent assay for detecting avian leukosis-sarcoma viruses.
about
A charged second-site mutation in the fusion peptide rescues replication of a mutant avian sarcoma and leukosis virus lacking critical cysteine residues flanking the internal fusion domain.Endogenous expression of ASLV viral proteins in specific pathogen free chicken embryos: relevance for the developmental biology research fieldSoluble forms of the subgroup A avian leukosis virus [ALV(A)] receptor Tva significantly inhibit ALV(A) infection in vitro and in vivo.Identification of key residues in subgroup A avian leukosis virus envelope determining receptor binding affinity and infectivity of cells expressing chicken or quail Tva receptor.The receptor for the subgroup C avian sarcoma and leukosis viruses, Tvc, is related to mammalian butyrophilins, members of the immunoglobulin superfamily.Activated BRAF induces gliomas in mice when combined with Ink4a/Arf loss or Akt activation.Targeted delivery of NRASQ61R and Cre-recombinase to post-natal melanocytes induces melanoma in Ink4a/Arflox/lox mice.Two different molecular defects in the Tva receptor gene explain the resistance of two tvar lines of chickens to infection by subgroup A avian sarcoma and leukosis viruses.Akt signaling is required for glioblastoma maintenance in vivoThe BRAF kinase domain promotes the development of gliomas in vivo.Ink4a/Arf loss promotes tumor recurrence following Ras inhibition.Efficient method to optimize antibodies using avian leukosis virus display and eukaryotic cellsAppropriate in vivo expression of a muscle-specific promoter by using avian retroviral vectors for gene transfer [corrected]Selective shedding and congenital transmission of endogenous avian leukosis viruses.Long terminal repeat (LTR) sequences, env, and a region near the 5' LTR influence the pathogenic potential of recombinants between Rous-associated virus types 0 and 1.Influence of env and long terminal repeat sequences on the tissue tropism of avian leukosis viruses.Embryonic infection with the endogenous avian leukosis virus Rous-associated virus-0 alters responses to exogenous avian leukosis virus infection.Efficient subgroup C avian sarcoma and leukosis virus receptor activity requires the IgV domain of the Tvc receptor and proper display on the cell membraneAkt signaling accelerates tumor recurrence following ras inhibition in the context of ink4a/arf loss.Assessing the roles of endogenous retrovirus EAV-HP in avian leukosis virus subgroup J emergence and tolerance.Evolutionary pressure of a receptor competitor selects different subgroup a avian leukosis virus escape variants with altered receptor interactions.Cloning, sequencing, and functional analysis of a Marek's disease virus origin of DNA replication.Delivery of short hairpin RNA sequences by using a replication-competent avian retroviral vector.Model of the TVA receptor determinants required for efficient infection by subgroup A avian sarcoma and leukosis viruses.
P2860
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P2860
An enzyme-linked immunosorbent assay for detecting avian leukosis-sarcoma viruses.
description
1979 nî lūn-bûn
@nan
1979年の論文
@ja
1979年論文
@yue
1979年論文
@zh-hant
1979年論文
@zh-hk
1979年論文
@zh-mo
1979年論文
@zh-tw
1979年论文
@wuu
1979年论文
@zh
1979年论文
@zh-cn
name
An enzyme-linked immunosorbent assay for detecting avian leukosis-sarcoma viruses.
@en
type
label
An enzyme-linked immunosorbent assay for detecting avian leukosis-sarcoma viruses.
@en
prefLabel
An enzyme-linked immunosorbent assay for detecting avian leukosis-sarcoma viruses.
@en
P2093
P1433
P1476
An enzyme-linked immunosorbent assay for detecting avian leukosis-sarcoma viruses.
@en
P2093
P304
P577
1979-07-01T00:00:00Z