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Endothelial transmigration by Trypanosoma cruziCurrent understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas diseaseEarly double-negative thymocyte export in Trypanosoma cruzi infection is restricted by sphingosine receptors and associated with human chagas disease.Caspase-1/ASC inflammasome-mediated activation of IL-1β-ROS-NF-κB pathway for control of Trypanosoma cruzi replication and survival is dispensable in NLRP3-/- macrophages.Amblyomma americanum tick calreticulin binds C1q but does not inhibit activation of the classical complement cascade.Deep sequencing of the Trypanosoma cruzi GP63 surface proteases reveals diversity and diversifying selection among chronic and congenital Chagas disease patients.P47phox-/- mice are compromised in expansion and activation of CD8+ T cells and susceptible to Trypanosoma cruzi infection.A Two-Component DNA-Prime/Protein-Boost Vaccination Strategy for Eliciting Long-Term, Protective T Cell Immunity against Trypanosoma cruziGenetic Susceptibility to Cardiac and Digestive Clinical Forms of Chronic Chagas Disease: Involvement of the CCR5 59029 A/G Polymorphism.Phenotypic Features of Circulating Leukocytes from Non-human Primates Naturally Infected with Trypanosoma cruzi Resemble the Major Immunological Findings Observed in Human Chagas Disease.ADENOSINE DEAMINASE ACTIVITY AND SERUM C-REACTIVE PROTEIN AS PROGNOSTIC MARKERS OF CHAGAS DISEASE SEVERITY.Developments in the management of Chagas cardiomyopathy.Pupillary Light Reflexes are Associated with Autonomic Dysfunction in Bolivian Diabetics But Not Chagas Disease Patients.The absence of myocardial calcium-independent phospholipase A2γ results in impaired prostaglandin E2 production and decreased survival in mice with acute Trypanosoma cruzi infection.Accelerating the development of a therapeutic vaccine for human Chagas disease: rationale and prospectsChemotherapeutic efficacy of phosphodiesterase inhibitors in chagasic cardiomyopathy.Membrane traffic and synaptic cross-talk during host cell entry by Trypanosoma cruzi.Autoimmune pathogenesis of Chagas heart disease: looking back, looking ahead.Sanitation for all: the global opportunity to increase transgenerational health gains and better understand the link between NCDs and NTDs, a scoping reviewVaccine-linked chemotherapy improves benznidazole efficacy for acute Chagas disease.New Insights into the Immunobiology of Mononuclear Phagocytic Cells and Their Relevance to the Pathogenesis of Cardiovascular Diseases.Pentose Phosphate Shunt Modulates Reactive Oxygen Species and Nitric Oxide Production Controlling Trypanosoma cruzi in Macrophages.PARP1 depletion improves mitochondrial and heart function in Chagas disease: Effects on POLG dependent mtDNA maintenance.
P2860
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P2860
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Pathogenesis of Chagas disease: time to move on.
@en
type
label
Pathogenesis of Chagas disease: time to move on.
@en
prefLabel
Pathogenesis of Chagas disease: time to move on.
@en
P2093
P2860
P356
P1476
Pathogenesis of Chagas disease: time to move on
@en
P2093
Fabiana S Machado
Fatima Brant
Lisia Esper
Mauro M Teixeira
P2860
P304
P356
10.2741/495
P577
2012-01-01T00:00:00Z