Capacitative Ca2+ entry is involved in regulating soluble amyloid precursor protein (sAPPalpha) release mediated by muscarinic acetylcholine receptor activation in neuroblastoma SH-SY5Y cells.
about
Hyperammonia induces specific liver injury through an intrinsic Ca2+-independent apoptosis pathwayOral Administration of Gintonin Attenuates Cholinergic Impairments by Scopolamine, Amyloid-β Protein, and Mouse Model of Alzheimer's Disease.TRPC6 specifically interacts with APP to inhibit its cleavage by γ-secretase and reduce Aβ production.Translationally Controlled Tumor Protein Stimulates Dopamine Release from PC12 Cells via Ca2+-Independent Phospholipase A₂ Pathways.TRPC Channels: Prominent Candidates of Underlying Mechanism in Neuropsychiatric Diseases.PKC links Gq-coupled receptors to DAT-mediated dopamine release.Nuclear factor-kappaB activated by capacitative Ca2+ entry enhances muscarinic receptor-mediated soluble amyloid precursor protein (sAPPalpha) release in SH-SY5Y cells.TRPC Channels and Alzheimer's Disease.
P2860
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P2860
Capacitative Ca2+ entry is involved in regulating soluble amyloid precursor protein (sAPPalpha) release mediated by muscarinic acetylcholine receptor activation in neuroblastoma SH-SY5Y cells.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Capacitative Ca2+ entry is inv ...... n neuroblastoma SH-SY5Y cells.
@en
type
label
Capacitative Ca2+ entry is inv ...... n neuroblastoma SH-SY5Y cells.
@en
prefLabel
Capacitative Ca2+ entry is inv ...... n neuroblastoma SH-SY5Y cells.
@en
P2093
P2860
P1476
Capacitative Ca2+ entry is inv ...... n neuroblastoma SH-SY5Y cells.
@en
P2093
Eun-Jihn Roh
Hwa-Jung Kim
Ji-Eun Jung
Jin Hyoung Kim
Sinkyu Choi
P2860
P304
P356
10.1111/J.1471-4159.2006.03734.X
P407
P577
2006-03-08T00:00:00Z