The dioxin receptor is silenced by promoter hypermethylation in human acute lymphoblastic leukemia through inhibition of Sp1 binding.
about
The aryl hydrocarbon receptor has an important role in the regulation of hematopoiesis: implications for benzene-induced hematopoietic toxicityEpigenetic regulation of human alpha1d-adrenergic receptor gene expression: a role for DNA methylation in Sp1-dependent regulationNew Trends in Aryl Hydrocarbon Receptor BiologyEpigenomics of leukemia: from mechanisms to therapeutic applicationsThe evolving role of the aryl hydrocarbon receptor (AHR) in the normophysiology of hematopoiesisGenome-wide B1 retrotransposon binds the transcription factors dioxin receptor and Slug and regulates gene expression in vivoNonadditive effects of PAHs on Early Vertebrate Development: mechanisms and implications for risk assessmentDifferential expression and role of hyperglycemia induced oxidative stress in epigenetic regulation of β1, β2 and β3-adrenergic receptors in retinal endothelial cellsOverexpression of Cu/Zn-superoxide dismutase and/or catalase accelerates benzo(a)pyrene detoxification by upregulation of the aryl hydrocarbon receptor in mouse endothelial cells.The aryl hydrocarbon receptor: regulation of hematopoiesis and involvement in the progression of blood diseases.Identification of methylated genes associated with aggressive clinicopathological features in mantle cell lymphoma.The aryl hydrocarbon receptor functions as a tumor suppressor of liver carcinogenesis.In silico discovery of novel transcription factors regulated by mTOR-pathway activities.Role of DNA methylation of AHR1 and AHR2 promoters in differential sensitivity to PCBs in Atlantic Killifish, Fundulus heteroclitus.Aryl hydrocarbon receptor-null allele mice have hematopoietic stem/progenitor cells with abnormal characteristics and functions.Development of a unique epigenetic signature during in vivo Th17 differentiation.Developmental exposure to 2,3,7,8 tetrachlorodibenzo-p-dioxin attenuates later-life Notch1-mediated T cell development and leukemogenesis.DNA Methylation in the Exon 1 Region and Complex Regulation of Twist1 Expression in Gastric Cancer CellsAHR promoter variant modulates its transcription and downstream effectors by allele-specific AHR-SP1 interaction functioning as a genetic marker for vitiligoThe aryl hydrocarbon receptor pathway: a key component of the microRNA-mediated AML signalisome.p23 co-chaperone protects the aryl hydrocarbon receptor from degradation in mouse and human cell lines.Potential roles of microRNA-29a in the molecular pathophysiology of T-cell acute lymphoblastic leukemia.The aryl hydrocarbon receptor, more than a xenobiotic-interacting protein.Treatment of mice with the Ah receptor agonist and human carcinogen dioxin results in altered numbers and function of hematopoietic stem cells.The dioxin receptor regulates the constitutive expression of the vav3 proto-oncogene and modulates cell shape and adhesion.The aryl hydrocarbon receptor has a normal function in the regulation of hematopoietic and other stem/progenitor cell populations.Discovery and characterization of Isofistularin-3, a marine brominated alkaloid, as a new DNA demethylating agent inducing cell cycle arrest and sensitization to TRAIL in cancer cells.The aryl hydrocarbon receptor cross-talks with multiple signal transduction pathways.DNA methylation and nucleosome occupancy regulate the cancer germline antigen gene MAGEA11.Loss of aryl hydrocarbon receptor promotes gene changes associated with premature hematopoietic stem cell exhaustion and development of a myeloproliferative disorder in aging mice.Genetic and epigenetic regulation of AHR gene expression in MCF-7 breast cancer cells: role of the proximal promoter GC-rich region.Epigenetic regulation of drug processing genes.The Ah receptor in stem cell cycling, regulation, and quiescenceSuppression of DPYD expression in RKO cells via DNA methylation in the regulatory region of the DPYD promoter: a potentially important epigenetic mechanism regulating DPYD expression.Aryl hydrocarbon receptor gene transitions (c.-742C>T; c.1661G>A) and idiopathic male infertility: a case-control study with in silico and meta-analysis.Aryl hydrocarbon receptor (AHR) is a potential tumour suppressor in pituitary adenomas.Dioxin receptor regulates aldehyde dehydrogenase to block melanoma tumorigenesis and metastasis.BCL6--regulated by AhR/ARNT and wild-type MEF2B--drives expression of germinal center markers MYBL1 and LMO2.The dioxin receptor has tumor suppressor activity in melanoma growth and metastasis.Dioxin receptor expression inhibits basal and transforming growth factor β-induced epithelial-to-mesenchymal transition.
P2860
Q24612123-ADD5FD51-F547-4199-A6CB-A2853B1091F9Q24646956-CB9AF702-BF11-4F79-B2F0-DDAC18AFC25CQ26744482-76E6A949-0F8C-43F6-8BF6-0D59F20E2853Q26823403-BC83B032-0956-41A5-AFCA-C930A8971497Q27025264-001FD437-4B50-447A-BC9E-391685D94AE5Q28510627-8FA16009-6F39-4E9D-AEF5-C7D5F64F3F43Q28751799-2DD4F5CB-E942-405C-9CC6-4704A53E076DQ33729972-D3E55DC7-9BDA-4981-A68D-C8D9C83ED615Q33758214-CCC38711-3B9C-4F27-89E9-BE5729C6F331Q33759140-64CE82AD-8F46-43E0-A823-8DBBDD0F58E3Q33908446-57EE6430-3068-4A3A-9708-EA5051C1C240Q34128053-C565F11B-9D2F-4E81-9988-7532DE066417Q34391073-88EFE9F2-2213-446F-BD0A-9F4B7E010138Q34483990-F280265F-309E-48E4-96EC-3F8C3EA55970Q35084108-6C18AEFF-3F16-4BB5-8047-4CE687179A4AQ35089039-80788DBB-2903-4976-89E7-DC6FCF14CDC5Q35090915-9CCF65CE-7B8D-4CE6-AA49-6217C935326DQ35875807-3F7376B7-FE7B-4BA3-9BE3-7AE01FA992B4Q36060628-21E8A99D-E3B7-48B8-B79E-EA08F751815FQ36067630-62AD2118-76B6-4BE0-BA3E-4E6002942101Q36161914-13632CAA-DE0B-4768-8CF9-B6E454A71218Q36262865-5CA92DA9-F555-4183-B1C9-9783FFAB59A7Q36782397-9382E219-0140-4017-A423-4D0B504D2F21Q37090414-D859CFB3-CC2D-4F1A-A690-A9419CD121EFQ37127317-CDBA8E1C-3B5E-482D-A149-968945C56227Q37150677-BD212F5C-7CCA-4BB0-AD0B-214F10E8CAE2Q37269205-C3C36DB5-2898-4AC5-8555-A1F9ACC7D302Q37279436-AA8EF0F6-B62B-4EF6-8045-AAB5A3600533Q37445288-0A9C2BB4-F3CD-4ED1-8554-253B384D6DEEQ37460084-B5BC985C-B163-4EFB-9C3D-4F5E0F677CBDQ37662171-EF3AF886-8834-40BC-80AC-BFA610D15446Q37865530-662988AD-C513-47E2-A9F6-60F5C064B9BEQ38185125-D250BE1C-61CB-40A0-A05D-18FE06BF6444Q38300283-59AEEFF5-51BB-4C42-B715-0E3A03E8F84CQ38677173-784DF85E-074C-4FE8-B96A-D4E0A5B2AAFCQ38709661-07A8DAD7-E5F6-4535-982A-5E7268C6882FQ38846315-3ED763F0-E2AC-49AE-9657-AACAA418FF53Q38900281-2439F419-C448-490C-91E5-7246871AA254Q39127681-AA4D0A79-21D7-4FFD-852C-908FCFB4F101Q39199510-26BF4260-123E-4D82-8151-A3B6E89E3627
P2860
The dioxin receptor is silenced by promoter hypermethylation in human acute lymphoblastic leukemia through inhibition of Sp1 binding.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
The dioxin receptor is silence ...... ugh inhibition of Sp1 binding.
@en
type
label
The dioxin receptor is silence ...... ugh inhibition of Sp1 binding.
@en
prefLabel
The dioxin receptor is silence ...... ugh inhibition of Sp1 binding.
@en
P2093
P50
P356
P1433
P1476
The dioxin receptor is silence ...... ough inhibition of Sp1 binding
@en
P2093
S Mulero-Navarro
P304
P356
10.1093/CARCIN/BGI344
P407
P577
2006-01-12T00:00:00Z