Smad2, Smad3 and Smad4 cooperate with Sp1 to induce p15(Ink4B) transcription in response to TGF-beta.
about
The transforming activity of Ski and SnoN is dependent on their ability to repress the activity of Smad proteinsZNF580, a novel C2H2 zinc-finger transcription factor, interacts with the TGF-β signal molecule Smad2Genome analysis identifies the p15ink4b tumor suppressor as a direct target of the ZNF217/CoREST complexNuclear export of Smad2 and Smad3 by RanBP3 facilitates termination of TGF-beta signalingIdentification of novel Smad2 and Smad3 associated proteins in response to TGF-beta1Homeodomain protein DLX4 counteracts key transcriptional control mechanisms of the TGF-β cytostatic program and blocks the antiproliferative effect of TGF-βIdentification of mZnf8, a mouse Krüppel-like transcriptional repressor, as a novel nuclear interaction partner of Smad1.Critical regulation of TGFbeta signaling by Hsp90Erbin inhibits transforming growth factor beta signaling through a novel Smad-interacting domainPKCalpha mediates TGFbeta-induced growth inhibition of human keratinocytes via phosphorylation of S100C/A11TGF-beta1 induction of the adenine nucleotide translocator 1 in astrocytes occurs through Smads and Sp1 transcription factorsRETRACTED: TGFβ1 signaling via αVβ6 integrinA critical role for the Sp1-binding sites in the transforming growth factor-beta-mediated inhibition of lipoprotein lipase gene expression in macrophagesSp1 and Smad proteins cooperate to mediate transforming growth factor-beta 1-induced alpha 2(I) collagen expression in human glomerular mesangial cellsActivation of transforming growth factor-beta signaling by SUMO-1 modification of tumor suppressor Smad4/DPC4Identification of a gadd45beta 3' enhancer that mediates SMAD3- and SMAD4-dependent transcriptional induction by transforming growth factor betaFLRG, an activin-binding protein, is a new target of TGFbeta transcription activation through Smad proteinsBoth Max and TFE3 cooperate with Smad proteins to bind the plasminogen activator inhibitor-1 promoter, but they have opposite effects on transcriptional activityRepression of bone morphogenetic protein and activin-inducible transcription by Evi-1Identification of binding sites of EVI1 in mammalian cellsIdentification and characterization of a novel gene, Mcpr1, and its possible function in the proliferation of embryonic palatal mesenchymal cellsThe anaphase-promoting complex coordinates initiation of lens differentiationArf induction by Tgfβ is influenced by Sp1 and C/ebpβ in opposing directionsLumican binds ALK5 to promote epithelium wound healingNuclear Export of Smads by RanBP3L Regulates Bone Morphogenetic Protein Signaling and Mesenchymal Stem Cell DifferentiationSmad4 dependency defines two classes of transforming growth factor {beta} (TGF-{beta}) target genes and distinguishes TGF-{beta}-induced epithelial-mesenchymal transition from its antiproliferative and migratory responses.Association between endogenous gene expression and growth regulation induced by TGF-beta1 in human gastric cancer cellsSox17 promotes cell cycle progression and inhibits TGF-beta/Smad3 signaling to initiate progenitor cell behavior in the respiratory epithelium.Pin1 promotes transforming growth factor-beta-induced migration and invasion.Specific control of BMP signaling and mesenchymal differentiation by cytoplasmic phosphatase PPM1H.Nullifying the CDKN2AB locus promotes mutant K-ras lung tumorigenesis.The BMP pathway acts to directly regulate Tbx20 in the developing heart.Requirement of a dynein light chain in TGFbeta/Smad3 signaling.SMAD4 mutations in colorectal cancer probably occur before chromosomal instability, but after divergence of the microsatellite instability pathwaySp1 and krüppel-like factor family of transcription factors in cell growth regulation and cancer.Bifurcated converging pathways for high Ca2+- and TGFbeta-induced inhibition of growth of normal human keratinocytesDEC1 is a downstream target of TGF-beta with sequence-specific transcriptional repressor activities.The endogenous ratio of Smad2 and Smad3 influences the cytostatic function of Smad3.TGF-beta receptor-activated p38 MAP kinase mediates Smad-independent TGF-beta responsesIntegrin αvβ3 drives slug activation and stemness in the pregnant and neoplastic mammary gland.
P2860
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P2860
Smad2, Smad3 and Smad4 cooperate with Sp1 to induce p15(Ink4B) transcription in response to TGF-beta.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Smad2, Smad3 and Smad4 coopera ...... ption in response to TGF-beta.
@en
type
label
Smad2, Smad3 and Smad4 coopera ...... ption in response to TGF-beta.
@en
prefLabel
Smad2, Smad3 and Smad4 coopera ...... ption in response to TGF-beta.
@en
P2093
P2860
P356
P1433
P1476
Smad2, Smad3 and Smad4 coopera ...... ption in response to TGF-beta.
@en
P2093
P2860
P304
P356
10.1093/EMBOJ/19.19.5178
P407
P577
2000-10-01T00:00:00Z