Tumor necrosis factor-alpha triggers mucus production in airway epithelium through an IkappaB kinase beta-dependent mechanism. - Wikidata - awgldk - TriplyDB

Tumor necrosis factor-alpha triggers mucus production in airway epithelium through an IkappaB kinase beta-dependent mechanism.

Tumor necrosis factor-alpha triggers mucus production in airway epithelium through an IkappaB kinase beta-dependent mechanism.

http://www.wikidata.org/entity/Q40381385

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S. mansoni bolsters anti-viral immunity in the murine respiratory tract Cellular crosstalk between airway epithelial and endothelial cells regulates barrier functions during exposure to double-stranded RNA Bergamot (Citrus bergamia Risso) fruit extracts and identified components alter expression of interleukin 8 gene in cystic fibrosis bronchial epithelial cell lines.Proteomics of buccal cavity mucus in female tilapia fish (Oreochromis spp.): a comparison between parental and non-parental fish.Indacaterol inhibits tumor cell invasiveness and MMP-9 expression by suppressing IKK/NF-κB activation.P. aeruginosa lipopolysaccharide-induced MUC5AC and CLCA3 expression is partly through Duox1 in vitro and in vivo.The tumor necrosis factor α (-308 A/G) polymorphism is associated with cystic fibrosis in Mexican patients.Dry powder formulation of simvastatin.Biological Effects of Simvastatin Formulated as pMDI on Pulmonary Epithelial Cells.Role of the Arylhydrocarbon Receptor (AhR) in the Pathology of Asthma and COPD.PPARγ as a Potential Target to Treat Airway Mucus Hypersecretion in Chronic Airway Inflammatory Diseases.Decrease in airway mucous gene expression caused by treatment with anti-tumor necrosis factor alpha in a murine model of allergic asthma.Regulation of airway mucin gene expression Regulation of airway MUC5AC expression by IL-1beta and IL-17A; the NF-kappaB paradigm.The Interaction between Respiratory Pathogens and Mucus.Transcriptome analyses in normal prostate epithelial cells exposed to low-dose cadmium: oncogenic and immunomodulations involving the action of tumor necrosis factor.Variants in mannose-binding lectin and tumour necrosis factor alpha affect survival in cystic fibrosis.Airway and lung pathology due to mucosal surface dehydration in {beta}-epithelial Na+ channel-overexpressing mice: role of TNF-{alpha} and IL-4R{alpha} signaling, influence of neonatal development, and limited efficacy of glucocorticoid treatment Using guinea pigs in studies relevant to asthma and COPD.Effects of intratracheal administration of nuclear factor-kappaB decoy oligodeoxynucleotides on long-term cigarette smoke-induced lung inflammation and pathology in mice.Could simvastatin be considered as a potential therapy for chronic lung diseases? A debate on the pros and cons.Induction of eosinophil apoptosis by hydrogen peroxide promotes the resolution of allergic inflammation S100A8, S100A9 and S100A12 activate airway epithelial cells to produce MUC5AC via extracellular signal-regulated kinase and nuclear factor-κB pathways.Pathobiology of cigarette smoke-induced chronic obstructive pulmonary disease.The signaling pathway involved in neutrophil elastase stimulated MUC1 transcription.Effect of pro-inflammatory stimuli on mucin expression and inhibition by secretory leucoprotease inhibitor.Peroxisome proliferator-activated receptor-gamma inhibits cigarette smoke solution-induced mucin production in human airway epithelial (NCI-H292) cells.Ribosomal protein S3 gene silencing protects against experimental allergic asthma.Distal airways are protected from goblet cell metaplasia by diminished expression of IL-13 signalling components.Maintenance of airway epithelium in acutely rejected orthotopic vascularized mouse lung transplants.α-Melanocyte-stimulating hormone inhibits tumor necrosis factor α-stimulated MUC5AC expression in human nasal epithelial cells.A novel antiinflammatory role for andrographolide in asthma via inhibition of the nuclear factor-kappaB pathway.Decreased expression of human aquaporin-5 correlated with mucus overproduction in airways of chronic obstructive pulmonary disease.Pinellia ternata Breitenbach attenuates ovalbumin-induced allergic airway inflammation and mucus secretion in a murine model of asthma.Glycogen Synthase Kinase-3β Inhibition Attenuates Asthma in Mice

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P2860

Tumor necrosis factor-alpha triggers mucus production in airway epithelium through an IkappaB kinase beta-dependent mechanism.

http://www.wikidata.org/entity/Q40381385

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

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Tumor necrosis factor-alpha tr ...... nase beta-dependent mechanism.

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Tumor necrosis factor-alpha tr ...... nase beta-dependent mechanism.

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Tumor necrosis factor-alpha tr ...... nase beta-dependent mechanism.

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Journal of Biological Chemistry

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Tumor necrosis factor-alpha tr ...... nase beta-dependent mechanism.

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P2093

Anne Marie King

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Anthony J Coyle

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Christopher C Fraser

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Dong Mei Zhang

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Jay Morgenstern

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José Carlos Gutiérrez-Ramos

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José M Lora

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Latika Singh

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Marie Keaveney

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Philip A Barker

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P2860

Dual oxidase 1-dependent MUC5AC mucin expression in cultured human airway epithelial cells

A simplified system for generating recombinant adenoviruses

Role of epidermal growth factor receptor activation in regulating mucin synthesis

Tumor necrosis factor-alpha +489G/A gene polymorphism is associated with chronic obstructive pulmonary disease

Cyclooxygenase-selective inhibition of prostanoid formation: transducing biochemical selectivity into clinical read-outs

TNF-R1 signaling: a beautiful pathway

The TNF and TNF receptor superfamilies: integrating mammalian biology

NF-kappaB regulation in the immune system

Human mucin gene MUC5AC: organization of its 5'-region and central repetitive region

Rapid activation of nuclear factor-kappaB in airway epithelium in a murine model of allergic airway inflammation

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10.1074/JBC.M507977200

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43

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280

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7637728

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16123045

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