Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids. - Wikidata - awgldk - TriplyDB

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

http://www.wikidata.org/entity/Q40384021

about

International Union of Basic and Clinical Pharmacology. LXXIX. Cannabinoid receptors and their ligands: beyond CB₁ and CB₂ Receptors and channels targeted by synthetic cannabinoid receptor agonists and antagonists.The endogenous brain constituent N-arachidonoyl L-serine is an activator of large conductance Ca2+-activated K+ channels Lipid regulation of BK channel function.The Role of BK Channels in Antiseizure Action of the CB1 Receptor Agonist ACEA in Maximal Electroshock and Pentylenetetrazole Models of Seizure in Mice Cytochrome P450 pathway contributes to methanandamide-induced vasorelaxation in rat aorta.Influence of methanandamide and CGRP on potassium currents in smooth muscle cells of small mesenteric arteries.Neurophysiological evidence for the presence of cannabinoid CB1 receptors in the laterodorsal tegmental nucleus.Dendritic calcium spikes are tunable triggers of cannabinoid release and short-term synaptic plasticity in cerebellar Purkinje neurons.Role of thromboxane A₂-activated nonselective cation channels in hypoxic pulmonary vasoconstriction of rat.

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P2860

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

http://www.wikidata.org/entity/Q40384021

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

@zh-hk

2005年論文

@zh-mo

2005年論文

@zh-tw

2005年论文

@wuu

2005年论文

@zh

2005年论文

@zh-cn

name

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

@en

type

label

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

@en

prefLabel

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

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P356

AJPCELL.00482.2004

P1433

American Journal of Physiology - Cell Physiology

P1476

Activation of large-conductance, Ca2+-activated K+ channels by cannabinoids.

@en

P2093

Hiroko Sade

http://www.w3.org/2001/XMLSchema#string

Katsuhiko Muraki

http://www.w3.org/2001/XMLSchema#string

Noriyuki Hatano

http://www.w3.org/2001/XMLSchema#string

Susumu Ohya

http://www.w3.org/2001/XMLSchema#string

Yuji Imaizumi

http://www.w3.org/2001/XMLSchema#string

P2860

Cloning and functional expression of two families of beta-subunits of the large conductance calcium-activated K+ channel

Determinant for beta-subunit regulation in high-conductance voltage-activated and Ca(2+)-sensitive K+ channels: an additional transmembrane region at the N terminus

Acute activation of Maxi-K channels (hSlo) by estradiol binding to the beta subunit

(R)-methanandamide: a chiral novel anandamide possessing higher potency and metabolic stability

Dihydroxyeicosatrienoic acids are potent activators of Ca(2+)-activated K(+) channels in isolated rat coronary arterial myocytes

The endocannabinoid anandamide is a direct and selective blocker of the background K(+) channel TASK-1.

Mechanisms of anandamide-induced vasorelaxation in rat isolated coronary arteries

Direct inhibition of T-type calcium channels by the endogenous cannabinoid anandamide

Vanilloid receptors on sensory nerves mediate the vasodilator action of anandamide

hKCNMB3 and hKCNMB4, cloning and characterization of two members of the large-conductance calcium-activated potassium channel beta subunit family.

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C77-86

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10.1152/AJPCELL.00482.2004

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1

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16107501

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