Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
about
The FUSE binding proteins FBP1 and FBP3 are potential c-myc regulators in renal, but not in prostate and bladder cancerPuralpha: a multifunctional single-stranded DNA- and RNA-binding proteinNuclear targeting determinants of the far upstream element binding protein, a c-myc transcription factorThe FUSE/FBP/FIR/TFIIH system is a molecular machine programming a pulse of c-myc expressionCooperative assembly of an hnRNP complex induced by a tissue-specific homolog of polypyrimidine tract binding proteinIdentification of human DNA helicase V with the far upstream element-binding proteinPATTERNFINDER: combined analysis of DNA regulatory sequences and double-helix stabilitySIDDBASE: a database containing the stress-induced DNA duplex destabilization (SIDD) profiles of complete microbial genomes.The FUSE Binding Protein Is a Cellular Factor Required for Efficient Replication of Hepatitis C VirusMolecular basis of sequence-specific single-stranded DNA recognition by KH domains: solution structure of a complex between hnRNP K KH3 and single-stranded DNADimerization of FIR upon FUSE DNA binding suggests a mechanism of c-myc inhibitionThe dynamic response of upstream DNA to transcription-generated torsional stressMYC on the path to cancerSuperhelical destabilization in regulatory regions of stress response genes.Candidate autoantigens identified by mass spectrometry in early rheumatoid arthritis are chaperones and citrullinated glycolytic enzymes.TFIIH operates through an expanded proximal promoter to fine-tune c-myc expression.Controlling the Master: Chromatin Dynamics at the MYC Promoter Integrate Developmental SignalingControlling gene expression by DNA mechanics: emerging insights and challenges.Role of far upstream element binding protein 1 in colonic epithelial disruption during dextran sulphate sodium-induced murine colitis.Theoretical analysis of the stress induced B-Z transition in superhelical DNA.Theoretical analysis of competing conformational transitions in superhelical DNA.The genome-wide distribution of non-B DNA motifs is shaped by operon structure and suggests the transcriptional importance of non-B DNA structures in Escherichia coliFuse binding protein antagonizes the transcription activity of tumor suppressor protein p53.Growth suppression by acute promyelocytic leukemia-associated protein PLZF is mediated by repression of c-myc expressionDifferential binding of replication proteins across the human c-myc replicator.FBPs are calibrated molecular tools to adjust gene expressionFUSE binding protein 1 interacts with untranslated regions of Japanese encephalitis virus RNA and negatively regulates viral replication.Identification of FUSE-binding protein 1 as a regulatory mRNA-binding protein that represses nucleophosmin translation.Far upstream element binding protein 1 activates translation of p27Kip1 mRNA through its internal ribosomal entry siteFar upstream element binding protein 1 binds the internal ribosomal entry site of enterovirus 71 and enhances viral translation and viral growth.A proteomic analysis of the functional effects of fatty acids in NIH 3T3 fibroblastsBathing in carbon dioxide-enriched water alters protein expression in keratinocytes of skin tissue in rats.Additive Promotion of Viral Internal Ribosome Entry Site-Mediated Translation by Far Upstream Element-Binding Protein 1 and an Enterovirus 71-Induced Cleavage Product.Activation of Cdc2 contributes to apoptosis in HPV E6 expressing human keratinocytes in response to therapeutic agentsUpregulation of Far Upstream Element-Binding Protein 1 (FUBP1) Promotes Tumor Proliferation and Tumorigenesis of Clear Cell Renal Cell Carcinoma.Far upstream element-binding protein 1 is a prognostic biomarker and promotes nasopharyngeal carcinoma progression.Overexpression of the far upstream element binding protein 1 in hepatocellular carcinoma is required for tumor growth.Hierarchical mechanisms build the DNA-binding specificity of FUSE binding protein.Far upstream element-binding protein 1 and RNA secondary structure both mediate second-step splicing repression.Far upstream element binding protein 1: a commander of transcription, translation and beyond
P2860
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P2860
Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
@en
type
label
Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
@en
prefLabel
Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
@en
P2093
P2860
P356
P1433
P1476
Loss of FBP function arrests cellular proliferation and extinguishes c-myc expression
@en
P2093
B O'Connell
C J Benham
P2860
P304
P356
10.1093/EMBOJ/19.5.1034
P407
P577
2000-03-01T00:00:00Z