Porphyromonas gingivalis lipopolysaccharide displays functionally diverse interactions with the innate host defense system.
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Expression of a Porphyromonas gingivalis lipid A palmitylacyltransferase in Escherichia coli yields a chimeric lipid A with altered ability to stimulate interleukin-8 secretion.Hemin-dependent modulation of the lipid A structure of Porphyromonas gingivalis lipopolysaccharide.Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation.Periodontitis, Porphyromonas, and the pathogenesis of rheumatoid arthritis.Porphyromonas gingivalis lipopolysaccharide weakly activates M1 and M2 polarized mouse macrophages but induces inflammatory cytokines.Porphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4.Porphyromonas gingivalis lipopolysaccharide antagonizes Escherichia coli lipopolysaccharide at toll-like receptor 4 in human endothelial cells.Hypoxia and P. gingivalis synergistically induce HIF-1 and NF-κB activation in PDL cells and periodontal diseases.Free lipid A isolated from Porphyromonas gingivalis lipopolysaccharide is contaminated with phosphorylated dihydroceramide lipids: recovery in diseased dental samples.Selective photoantisepsis.Serine dipeptide lipids of Porphyromonas gingivalis inhibit osteoblast differentiation: Relationship to Toll-like receptor 2Role of the innate immune system in host defence against bacterial infections: focus on the Toll-like receptors.Serine lipids of Porphyromonas gingivalis are human and mouse Toll-like receptor 2 ligandsPolybacterial challenge effects on cytokine/chemokine production by macrophages and dendritic cells.Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.Porphyromonas gingivalis Fimbria-Induced Expression of Inflammatory Cytokines and Cyclooxygenase-2 in Mouse Macrophages and Its Inhibition by the Bioactive Compounds Fibronectin and Melatonin.PKC, ERK/p38 MAP kinases and NF-κB targeted signalling play a role in the expression and release of IL-1β and CXCL8 in Porphyromonas gingivalis-infected THP1 cells.Gingipain of Porphyromonas gingivalis manipulates M1 macrophage polarization through C5a pathway.Regulation of protease-activated receptor-2 expression in gingival fibroblasts and Jurkat T cells by Porphyromonas gingivalis.Porphyromonas gingivalis lipopolysaccharide contains multiple lipid A species that functionally interact with both toll-like receptors 2 and 4.Differential activation of NF-kappaB and gene expression in oral epithelial cells by periodontal pathogens.HtpG, the Porphyromonas gingivalis HSP-90 homologue, induces the chemokine CXCL8 in human monocytic and microvascular vein endothelial cells.Interleukin-1alpha stimulation in monocytes by periodontal bacteria: antagonistic effects of Porphyromonas gingivalis.Porphyromonas gingivalis lipopolysaccharide lipid A heterogeneity: differential activities of tetra- and penta-acylated lipid A structures on E-selectin expression and TLR4 recognition.Toll-like receptor agonists Porphyromonas gingivalis LPS and CpG differentially regulate IL-10 competency and frequencies of mouse B10 cells.Impaired Porphyromonas gingivalis-Induced Tumor Necrosis Factor Production by Dendritic Cells Typifies Patients With Rheumatoid Arthritis.Immune response of macrophages from young and aged mice to the oral pathogenic bacterium Porphyromonas gingivalis.Role of MyD88-dependent and MyD88-independent signaling in Porphyromonas gingivalis-elicited macrophage foam cell formation.Porphyromonas gingivalis lipids inhibit osteoblastic differentiation and function.Scavenger receptor A is expressed by macrophages in response to Porphyromonas gingivalis, and participates in TNF-alpha expression.Nuclear factor-κB and p38 mitogen-activated protein kinase signaling pathways are critically involved in Porphyromonas gingivalis lipopolysaccharide induction of lipopolysaccharide-binding protein expression in human oral keratinocytes.Identification of an anti-inflammatory potential of Eriodictyon angustifolium compounds in human gingival fibroblasts.Different effects of Porphyromonas gingivalis lipopolysaccharide and TLR2 agonist Pam3CSK4 on the adhesion molecules expression in endothelial cells.Role of p38 mitogen-activated protein kinase pathway in Porphyromonas gingivalis lipopolysaccharide-induced VCAM-1 expression in human aortic endothelial cells.Phosphorylcholine mimics the effects of ES-62 on macrophages and dendritic cells.Cementoblast gene expression is regulated by Porphyromonas gingivalis lipopolysaccharide partially via toll-like receptor-4/MD-2.
P2860
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P2860
Porphyromonas gingivalis lipopolysaccharide displays functionally diverse interactions with the innate host defense system.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
type
label
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
prefLabel
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
P2093
P2860
P1476
Porphyromonas gingivalis lipop ...... he innate host defense system.
@en
P2093
Brian W Bainbridge
Richard P Darveau
Stephen R Coats
P2860
P356
10.1902/ANNALS.2002.7.1.29
P577
2002-12-01T00:00:00Z