Cell cycle arrest in response to DNA damage: lessons from yeast.
about
SIR functions are required for the toleration of an unrepaired double-strand break in a dispensable yeast chromosome.Requirement for end-joining and checkpoint functions, but not RAD52-mediated recombination, after EcoRI endonuclease cleavage of Saccharomyces cerevisiae DNA.Repair of endonuclease-induced double-strand breaks in Saccharomyces cerevisiae: essential role for genes associated with nonhomologous end-joining.DNA damage-inducible and RAD52-independent repair of DNA double-strand breaks in Saccharomyces cerevisiae.Cloning and characterization of RAD17, a gene controlling cell cycle responses to DNA damage in Saccharomyces cerevisiae.DNA damage checkpoints are involved in postreplication repair.The Saccharomyces cerevisiae Ku autoantigen homologue affects radiosensitivity only in the absence of homologous recombination.Identification of Myc-mediated death response pathways by microarray analysis.
P2860
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P2860
Cell cycle arrest in response to DNA damage: lessons from yeast.
description
1995 nî lūn-bûn
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1995年の論文
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1995年学术文章
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1995年学术文章
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1995年学术文章
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1995年学术文章
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1995年学术文章
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1995年學術文章
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1995年學術文章
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1995年學術文章
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name
Cell cycle arrest in response to DNA damage: lessons from yeast.
@en
type
label
Cell cycle arrest in response to DNA damage: lessons from yeast.
@en
prefLabel
Cell cycle arrest in response to DNA damage: lessons from yeast.
@en
P1433
P1476
Cell cycle arrest in response to DNA damage: lessons from yeast.
@en
P2093
P356
10.1016/0921-8777(95)00023-D
P407
P577
1995-09-01T00:00:00Z