STAT3 is a potential modulator of HIF-1-mediated VEGF expression in human renal carcinoma cells. - Wikidata - awgldk - TriplyDB

STAT3 is a potential modulator of HIF-1-mediated VEGF expression in human renal carcinoma cells.

STAT3 is a potential modulator of HIF-1-mediated VEGF expression in human renal carcinoma cells.

http://www.wikidata.org/entity/Q40416582

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Effect of hypoxia on the expression of αB-crystallin in head and neck squamous cell carcinoma Mammalian target of rapamycin up-regulation of pyruvate kinase isoenzyme type M2 is critical for aerobic glycolysis and tumor growth Neutrophils responsive to endogenous IFN-beta regulate tumor angiogenesis and growth in a mouse tumor model STAT3 inhibits the degradation of HIF-1alpha by pVHL-mediated ubiquitination Immunological Evasion in Glioblastoma The Role of the Transcriptional Regulation of Stromal Cells in Chronic Inflammation Methylsulfonylmethane suppresses breast cancer growth by down-regulating STAT3 and STAT5b pathways Hwanggeumchal sorghum induces cell cycle arrest, and suppresses tumor growth and metastasis through Jak2/STAT pathways in breast cancer xenografts The role of STAT3 in autophagy Molecular targets of isothiocyanates in cancer: recent advances Hyperglycaemia exacerbates choroidal neovascularisation in mice via the oxidative stress-induced activation of STAT3 signalling in RPE cells JSI-124 suppresses invasion and angiogenesis of glioblastoma cells in vitro Tyk2 and Stat3 regulate brown adipose tissue differentiation and obesity TCGA data and patient-derived orthotopic xenografts highlight pancreatic cancer-associated angiogenesis Structurally modified curcumin analogs inhibit STAT3 phosphorylation and promote apoptosis of human renal cell carcinoma and melanoma cell lines.STAT3 or USF2 contributes to HIF target gene specificity.Analysis of the retinal gene expression profile after hypoxic preconditioning identifies candidate genes for neuroprotection.Interleukin-10 promotes pathological angiogenesis by regulating macrophage response to hypoxia during development.Hypoxic repression of endothelial nitric-oxide synthase transcription is coupled with eviction of promoter histones Stat3 activation is required for the growth of U87 cell-derived tumours in mice The SWI/SNF Protein PBRM1 Restrains VHL-Loss-Driven Clear Cell Renal Cell Carcinoma.VEGF-A Stimulates STAT3 Activity via Nitrosylation of Myocardin to Regulate the Expression of Vascular Smooth Muscle Cell Differentiation Markers.STAT3, p-STAT3 and HIF-1α are associated with vasculogenic mimicry and impact on survival in gastric adenocarcinoma Hypoxia potentiates glioma-mediated immunosuppression STAT3 inhibitor WP1066 as a novel therapeutic agent for renal cell carcinoma.Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension.Hypoxia induces chemoresistance in ovarian cancer cells by activation of signal transducer and activator of transcription 3.Suppression of STAT3 and HIF-1 alpha mediates anti-angiogenic activity of betulinic acid in hypoxic PC-3 prostate cancer cells.YLT192, a novel, orally active bioavailable inhibitor of VEGFR2 signaling with potent antiangiogenic activity and antitumor efficacy in preclinical models Benzyl isothiocyanate suppresses pancreatic tumor angiogenesis and invasion by inhibiting HIF-α/VEGF/Rho-GTPases: pivotal role of STAT-3.Signal transducer and activator of transcription 3-stimulated hypoxia inducible factor-1alpha mediates estrogen receptor-alpha-induced mesenchymal stem cell vascular endothelial growth factor production Diindolylmethane suppresses ovarian cancer growth and potentiates the effect of cisplatin in tumor mouse model by targeting signal transducer and activator of transcription 3 (STAT3).Receptor "hijacking" by malignant glioma cells: a tactic for tumor progression Association of pSTAT3-VEGF signaling pathway with peritumoral edema in newly diagnosed glioblastoma: an immunohistochemical study.Hypoxia induces trimethylated H3 lysine 4 by inhibition of JARID1A demethylase Activation of the AT1R/HIF-1 α /ACE axis mediates angiotensin II-induced VEGF synthesis in mesenchymal stem cells.Peroxiredoxin 1 stimulates endothelial cell expression of VEGF via TLR4 dependent activation of HIF-1α.Hypoxia-inducible factor-1alpha enhances haptoglobin gene expression by improving binding of STAT3 to the promoter.Cathepsin B and uPAR knockdown inhibits tumor-induced angiogenesis by modulating VEGF expression in glioma (-)-Epigallocatechin-3-gallate inhibits VEGF expression induced by IL-6 via Stat3 in gastric cancer.

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P2860

STAT3 is a potential modulator of HIF-1-mediated VEGF expression in human renal carcinoma cells.

http://www.wikidata.org/entity/Q40416582

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

STAT3 is a potential modulator ...... n human renal carcinoma cells.

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type

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STAT3 is a potential modulator ...... n human renal carcinoma cells.

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STAT3 is a potential modulator ...... n human renal carcinoma cells.

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Doo Hyun Chung

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Hyun Gyu Lee

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Ik Hyun Cho

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Jong-Wan Park

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Joo Eun Jung

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Jung Weon Lee

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Myung-Hee Chung

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Sang-Kyu Ye

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Seongwon Choi

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Sun-Hee Yoon

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1296-1298

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10.1096/FJ.04-3099FJE

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19

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15919761

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