Ethanol alters cellular activation and CD14 partitioning in lipid rafts.
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Ethanol causes the redistribution of L1 cell adhesion molecule in lipid raftsEthanol inhibits L1 cell adhesion molecule activation of mitogen-activated protein kinasesEthanol/naltrexone interactions at the mu-opioid receptor. CLSM/FCS study in live cells.Ethanol inhibits LPS-induced signaling and modulates cytokine production in peritoneal macrophages in vivo in a model for binge drinking.Investigation of the role of TNF-α converting enzyme (TACE) in the inhibition of cell surface and soluble TNF-α production by acute ethanol exposureInnate immunity and inflammation in sepsis: mechanisms of suppressed host resistance in mice treated with ethanol in a binge-drinking modelRole of lipid rafts in liver health and diseaseNanoscale effects of ethanol and naltrexone on protein organization in the plasma membrane studied by photoactivated localization microscopy (PALM).Lysophosphatidic acid receptor 1 modulates lipopolysaccharide-induced inflammation in alveolar epithelial cells and murine lungs.Role of acute ethanol exposure and TLR4 in early events of sepsis in a mouse model.Down-regulation of Stathmin Is Required for the Phenotypic Changes and Classical Activation of Macrophages.Carbon monoxide differentially inhibits TLR signaling pathways by regulating ROS-induced trafficking of TLRs to lipid rafts.L1 cell adhesion molecule signaling is inhibited by ethanol in vivoExpanding research on the role of alcohol consumption and related risks in the prevention and treatment of HIV/AIDS.Exposure-dependent effects of ethanol on the innate immune system.Binge ethanol and liver: new molecular developmentsThe size of the unbranched aliphatic chain determines the immunomodulatory potency of short and long chain n-alkanolsIn vitro and in vivo models of acute alcohol exposure.Ethanol inhibits L1 cell adhesion molecule tyrosine phosphorylation and dephosphorylation and activation of pp60(src).A Comprehensive View of the Neurotoxicity Mechanisms of Cocaine and Ethanol.Ethanol suppresses LPS-induced Toll-like receptor 4 clustering, reorganization of the actin cytoskeleton, and associated TNF-alpha production.ELISA assays and alcohol: increasing carbon chain length can interfere with detection of cytokines.Ethanol inhibits lipid raft-mediated TCR signaling and IL-2 expression: potential mechanism of alcohol-induced immune suppression.Ethanol exposure suppresses bone marrow-derived dendritic cell inflammatory responses independent of TLR4 expression.L1 cell adhesion molecule promotes resistance to alcohol-induced silencing of growth cone responses to guidance cues.Involvement of Toll-like receptor 4 in acetaminophen hepatotoxicity.Ethanol affects the generation, cosignaling molecule expression, and function of plasmacytoid and myeloid dendritic cell subsets in vitro and in vivo.
P2860
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P2860
Ethanol alters cellular activation and CD14 partitioning in lipid rafts.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Ethanol alters cellular activation and CD14 partitioning in lipid rafts.
@en
type
label
Ethanol alters cellular activation and CD14 partitioning in lipid rafts.
@en
prefLabel
Ethanol alters cellular activation and CD14 partitioning in lipid rafts.
@en
P1476
Ethanol alters cellular activation and CD14 partitioning in lipid rafts
@en
P2093
Stephen B Pruett
P356
10.1016/J.BBRC.2005.04.088
P50
P577
2005-06-01T00:00:00Z