ER alpha-AHR-ARNT protein-protein interactions mediate estradiol-dependent transrepression of dioxin-inducible gene transcription.
about
Coactivator recruitment of AhR/ARNT1The aryl hydrocarbon receptor complex and the control of gene expressionGenetic modification of the association between peripubertal dioxin exposure and pubertal onset in a cohort of Russian boysZebrafish whole-adult-organism chemogenomics for large-scale predictive and discovery chemical biologyA TRIP230-retinoblastoma protein complex regulates hypoxia-inducible factor-1α-mediated transcription and cancer cell invasionAntagonism of aryl hydrocarbon receptor signaling by 6,2',4'-trimethoxyflavoneGenome-wide computational analysis of dioxin response element location and distribution in the human, mouse, and rat genomes.Dioxin and estrogen signaling in lung adenocarcinoma cells with different aryl hydrocarbon receptor/estrogen receptor α phenotypes.Development of a selective modulator of aryl hydrocarbon (Ah) receptor activity that exhibits anti-inflammatory properties.Distinct roles for aryl hydrocarbon receptor nuclear translocator and ah receptor in estrogen-mediated signaling in human cancer cell lines.Estrogen receptor expression is required for low-dose resveratrol-mediated repression of aryl hydrocarbon receptor activity.Defining molecular sensors to assess long-term effects of pesticides on carcinogenesis.Low levels of 3,3'-diindolylmethane activate estrogen receptor α and induce proliferation of breast cancer cells in the absence of estradiol.Xenobiotic metabolism, disposition, and regulation by receptors: from biochemical phenomenon to predictors of major toxicitiesAryl hydrocarbon receptor activation in lactotropes and gonadotropes interferes with estradiol-dependent and -independent preprolactin, glycoprotein alpha and luteinizing hormone beta gene expressionEstrogen receptor and aryl hydrocarbon receptor signaling pathwaysReactivation of estrogen receptor α by vorinostat sensitizes mesenchymal-like triple-negative breast cancer to aminoflavone, a ligand of the aryl hydrocarbon receptor.Growth of a human mammary tumor cell line is blocked by galangin, a naturally occurring bioflavonoid, and is accompanied by down-regulation of cyclins D3, E, and A.Identification of estrogen target genes during zebrafish embryonic development through transcriptomic analysisAryl hydrocarbon receptor ligands in cancer: friend and foe.Estrogen receptor α enhances the transcriptional activity of ETS-1 and promotes the proliferation, migration and invasion of neuroblastoma cell in a ligand dependent manner.Aryl hydrocarbon receptor modulation of estrogen receptor α-mediated gene regulation by a multimeric chromatin complex involving the two receptors and the coregulator RIP140.Endocrine disrupting chemicals: Multiple effects on testicular signaling and spermatogenesis.The impact of aryl hydrocarbon receptor signaling on matrix metabolism: implications for development and disease.The dioxin (aryl hydrocarbon) receptor as a model for adaptive responses of bHLH/PAS transcription factors.Cross-Talk in the Female Rat Mammary Gland: Influence of Aryl Hydrocarbon Receptor on Estrogen Receptor SignalingInflammatory signaling and aryl hydrocarbon receptor mediate synergistic induction of interleukin 6 in MCF-7 cells.Ah receptor binding to its cognate response element is required for dioxin-mediated toxicity.Role of the aryl hydrocarbon receptor in carcinogenesis and potential as a drug target.Regulation of constitutive and inducible AHR signaling: complex interactions involving the AHR repressorAHR-dependent misregulation of Wnt signaling disrupts tissue regeneration.A single gestational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin disrupts the adult uterine response to estradiol in mice.A truncated Ah receptor blocks the hypoxia and estrogen receptor signaling pathways: a viable approach for breast cancer treatment.An overview of the effects of dioxins and dioxin-like compounds on vertebrates, as documented in human and ecological epidemiologyHormonal regulation of CYP1A expression.Correlation between dioxin and endometriosis: an epigenetic route to unravel the pathogenesis of the disease.Transgenic overexpression of p23 induces spontaneous hydronephrosis in mice.Mechanisms and therapeutic prospects of polyphenols as modulators of the aryl hydrocarbon receptor.Effect of nonpersistent pesticides on estrogen receptor, androgen receptor, and aryl hydrocarbon receptor.Ah receptor antagonism represses head and neck tumor cell aggressive phenotype.
P2860
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P2860
ER alpha-AHR-ARNT protein-protein interactions mediate estradiol-dependent transrepression of dioxin-inducible gene transcription.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
ER alpha-AHR-ARNT protein-prot ...... -inducible gene transcription.
@en
type
label
ER alpha-AHR-ARNT protein-prot ...... -inducible gene transcription.
@en
prefLabel
ER alpha-AHR-ARNT protein-prot ...... -inducible gene transcription.
@en
P2860
P356
P1476
ER alpha-AHR-ARNT protein-prot ...... -inducible gene transcription.
@en
P2093
Gary H Perdew
Timothy V Beischlag
P2860
P304
21607-21611
P356
10.1074/JBC.C500090200
P407
P577
2005-04-18T00:00:00Z