about
The intracellular sensor NLRP3 mediates key innate and healing responses to influenza A virus via the regulation of caspase-1Balancing Immune Protection and Immune Pathology by CD8(+) T-Cell Responses to Influenza InfectionAn Epithelial Integrin Regulates the Amplitude of Protective Lung Interferon Responses against Multiple Respiratory PathogensEpitope-specific TCR repertoire diversity imparts no functional advantage on the CD8+ T cell response to cognate viral peptidesProtective Efficacy of Cross-Reactive CD8+ T Cells Recognising Mutant Viral Epitopes Depends on Peptide-MHC-I Structural Interactions and T Cell Activation ThresholdOseltamivir Prophylaxis Reduces Inflammation and Facilitates Establishment of Cross-Strain Protective T Cell Memory to Influenza VirusesEcological analysis of antigen-specific CTL repertoires defines the relationship between naive and immune T-cell populationsImplementing hospital-based surveillance for severe acute respiratory infections caused by influenza and other respiratory pathogens in New Zealand.Cytomegalovirus infection enhances the immune response to influenza.Cell-mediated protection in influenza infection.The kinase mTOR modulates the antibody response to provide cross-protective immunity to lethal infection with influenza virus.Human H7N9 and H5N1 influenza viruses differ in induction of cytokines and tissue tropism.Seasonal influenza vaccination is the strongest correlate of cross-reactive antibody responses in migratory bird handlers.Diverse heterologous primary infections radically alter immunodominance hierarchies and clinical outcomes following H7N9 influenza challenge in miceA comprehensive collection of systems biology data characterizing the host response to viral infection.TNF/iNOS-producing dendritic cells are the necessary evil of lethal influenza virus infection.Protective memory responses are modulated by priming events prior to challenge.Contemporary seasonal influenza A (H1N1) virus infection primes for a more robust response to split inactivated pandemic influenza A (H1N1) Virus vaccination in ferretsImmunity to seasonal and pandemic influenza A virusesEosinophils Promote Antiviral Immunity in Mice Infected with Influenza A VirusConstrained evolution drives limited influenza diversity.Trans-nodal migration of resident dendritic cells into medullary interfollicular regions initiates immunity to influenza vaccine.MR1-restricted mucosal-associated invariant T (MAIT) cells respond to mycobacterial vaccination and infection in nonhuman primatesPhysiological numbers of CD4+ T cells generate weak recall responses following influenza virus challengeConsequences of immunodominant epitope deletion for minor influenza virus-specific CD8+-T-cell responses.Membrane association of the CD3ε signaling domain is required for optimal T cell development and function.Primary CTL response magnitude in mice is determined by the extent of naive T cell recruitment and subsequent clonal expansionNucleotide oligomerization and binding domain 2-dependent dendritic cell activation is necessary for innate immunity and optimal CD8+ T Cell responses to influenza A virus infectionDangerous for ferrets: lethal for humans?Paired analysis of TCRα and TCRβ chains at the single-cell level in mice.An unexpected antibody response to an engineered influenza virus modifies CD8+ T cell responses.T Cell receptor clonotype influences epitope hierarchy in the CD8+ T cell response to respiratory syncytial virus infection.Gamma delta T cell reconstitution is associated with fewer infections and improved event-free survival after hematopoietic stem cell transplantation for pediatric leukemia.Differential host response, rather than early viral replication efficiency, correlates with pathogenicity caused by influenza viruses.Clonally related CD8+ T cells responsible for rapid population of both diffuse nasal-associated lymphoid tissue and lung after respiratory virus infectionDetection of antibodies against Turkey astrovirus in humans.T cell immunoglobulin and mucin protein-3 (Tim-3)/Galectin-9 interaction regulates influenza A virus-specific humoral and CD8 T-cell responses.Neonatal CD8 T-cell hierarchy is distinct from adults and is influenced by intrinsic T cell properties in respiratory syncytial virus infected mice.Immune biasing by helminth glycans.Recovery from severe H7N9 disease is associated with diverse response mechanisms dominated by CD8⁺ T cells.
P50
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P50
description
hulumtues
@sq
onderzoeker
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researcher
@en
հետազոտող
@hy
name
Paul G. Thomas
@ast
Paul G. Thomas
@en
Paul G. Thomas
@es
Paul G. Thomas
@nl
Paul G. Thomas
@sl
type
label
Paul G. Thomas
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Paul G. Thomas
@en
Paul G. Thomas
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Paul G. Thomas
@nl
Paul G. Thomas
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prefLabel
Paul G. Thomas
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Paul G. Thomas
@en
Paul G. Thomas
@es
Paul G. Thomas
@nl
Paul G. Thomas
@sl
P1053
H-1063-2018
P106
P1153
8264456400
P21
P31
P3829
P3835
paul-thomas17
P496
0000-0001-7955-0256