A strategy for establishing mode of action of chemical carcinogens as a guide for approaches to risk assessments.
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Use of mechanistic data in assessing human risks from exposure to particlesMarriage of a medium-term liver model to surrogate markers--a practical approach for risk and benefit assessment.Health risks associated with inhaled nasal toxicants.The significance of mouse liver tumor formation for carcinogenic risk assessment: results and conclusions from a survey of ten years of testing by the agrochemical industryEffect of vitamin E on hepatic cell proliferation and apoptosis in mice deficient in the p50 subunit of NF-κB after treatment with phenobarbital.Successful drug development despite adverse preclinical findings part 1: processes to address issues and most important findingsEffect of phenobarbital on hepatic cell proliferation and apoptosis in mice deficient in the p50 subunit of NF-kappaBToxicity and Carcinogenicity of Dichlorodiphenyltrichloroethane (DDT).Does GLP enhance the quality of toxicological evidence for regulatory decisions?Cell proliferation in nasal respiratory epithelium of people exposed to urban pollution.Differential display identified changes in mRNA levels in regenerating livers from chloroform-treated mice.A brief review of formaldehyde carcinogenesis in relation to rat nasal pathology and human health risk assessment.Long-term mutagenicity studies with chloroform and dimethylnitrosamine in female lacI transgenic B6C3F1 mice.Twenty-six-Week carcinogenicity study of chloroform in CB6F1 rasH2-transgenic mice.Interlaboratory comparison of short-term carcinogenicity studies using CB6F1-rasH2 transgenic mice.Contamination is a frequent confounding factor in toxicology studies with anthraquinone and related compounds.An autoradiographic study of cellular proliferaton, DNA synthesis and cell cycle variability in the rat liver caused by phenobarbital-induced oxidative stress: the protective role of melatonin.Differential display in rat livers treated for 13 weeks with phenobarbital implicates a role for metabolic and oxidative stress in nongenotoxic carcinogenicity.Mechanism of action of the nongenotoxic peroxisome proliferators: role of the peroxisome proliferator-activator receptor alpha.Toxicity profiles in mice treated with hepatotumorigenic and non-hepatotumorigenic triazole conazole fungicides: Propiconazole, triadimefon, and myclobutanil.
P2860
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P2860
A strategy for establishing mode of action of chemical carcinogens as a guide for approaches to risk assessments.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
A strategy for establishing mo ...... pproaches to risk assessments.
@en
type
label
A strategy for establishing mo ...... pproaches to risk assessments.
@en
prefLabel
A strategy for establishing mo ...... pproaches to risk assessments.
@en
P2093
P1433
P1476
A strategy for establishing mo ...... pproaches to risk assessments.
@en
P2093
Butterworth BE
Conolly RB
P304
P356
10.1016/0304-3835(95)03794-W
P407
P577
1995-06-01T00:00:00Z