The second loop of occludin is required for suppression of Raf1-induced tumor growth.
about
Identification of MarvelD3 as a tight junction-associated transmembrane protein of the occludin familyMarvelD3 couples tight junctions to the MEKK1-JNK pathway to regulate cell behavior and survivalTight Junctions and the Tumor MicroenvironmentTumor necrosis factor receptor-associated factor 4 is a dynamic tight junction-related shuttle protein involved in epithelium homeostasisIdentification and Analysis of Occludin Phosphosites: A Combined Mass Spectrometry and Bioinformatics ApproachPhysiology and function of the tight junction.Glycogen Synthase Kinase 3 (GSK-3) influences epithelial barrier function by regulating occludin, claudin-1 and E-cadherin expression.Occludin localizes to centrosomes and modifies mitotic entry.A key claudin extracellular loop domain is critical for epithelial barrier integrityTriggers and effectors of oxidative stress at blood-brain barrier level: relevance for brain ageing and neurodegenerationOccludin independently regulates permeability under hydrostatic pressure and cell division in retinal pigment epithelial cellsRole of the intestinal barrier in inflammatory bowel disease.Tight junction proteins: from barrier to tumorigenesis.Tight junctions and the modulation of barrier function in diseaseMeprin A impairs epithelial barrier function, enhances monocyte migration, and cleaves the tight junction protein occludin.Tight junctions: a barrier to the initiation and progression of breast cancer?Review: molecular pathogenesis of blood-brain barrier breakdown in acute brain injury.Regulation of epithelial proliferation by tight junction proteins.JAM-related proteins in mucosal homeostasis and inflammationIntestinal epithelial claudins: expression and regulation in homeostasis and inflammation.Occludin S490 Phosphorylation Regulates Vascular Endothelial Growth Factor-Induced Retinal Neovascularization.The second extracellular loop dictates Occludin-mediated HCV entryCo-culture based blood-brain barrier in vitro model, a tissue engineering approach using immortalized cell lines for drug transport study.Regulation of renal epithelial tight junctions by the von Hippel-Lindau tumor suppressor gene involves occludin and claudin 1 and is independent of E-cadherin.A novel splice variant of occludin deleted in exon 9 and its role in cell apoptosis and invasion.Occludin-mediated premature senescence is a fail-safe mechanism against tumorigenesis in breast carcinoma cells.Occludin S471 Phosphorylation Contributes to Epithelial Monolayer Maturation.Glucocorticoids induce transactivation of tight junction genes occludin and claudin-5 in retinal endothelial cells via a novel cis-element.
P2860
Q24293163-1D1830D3-7D55-4227-8410-0BC43A37BC80Q24338226-F4689CDE-BE2C-4B0C-B96A-861A8DF0A9ACQ26740056-FC0834EB-3EAF-4542-8726-75E7D81B10F9Q27438162-09310166-28A8-4107-B989-61F078DB5F3EQ27653352-5278FEF7-1A16-4A5C-9151-E275D50576D3Q28270069-34CD6610-F5AA-49CE-8CAC-E09B5E801D3FQ34284161-EA6A4434-94B9-4E6C-B774-0273AC866B55Q35182998-13575906-33BB-4801-8055-27026F569A14Q36512295-18E516EB-3FB6-439B-98AF-C0C77C1D64C6Q36711812-24DE46F2-CA76-47A1-AE7C-7044B0DEA73CQ36789765-E606B148-E31F-4072-A8A2-6D2D523685E6Q37058869-8707C431-3AC1-4EAD-BCC3-239D54568C34Q37118065-CF3EF5F6-2AE6-41EB-8CFF-AA2025E6CE4FQ37139446-9B279DD9-0A08-4B65-A47A-04C22E643E62Q37142513-2200525F-316B-4121-80B4-BA062A38493BQ37420725-F958AFF9-A8D5-4DB3-861B-F2E865095184Q37800133-1A760533-1E7A-45FB-92F6-9CA5FE44FCFCQ38021500-41D7BCA7-197B-41C8-B177-DD2754F950E2Q38199343-DA3931E5-12D0-43BE-833B-DBFA5D8137FFQ39298268-EA5C6930-4EDE-4451-BB72-4B6FDC011C67Q39595301-CE4D6257-78DA-466A-B03B-440A64D0A04EQ39658657-1CF1EAC6-06A0-4576-8F7E-D6E1F0984C02Q39676905-6BE050F9-C1A4-4927-8B03-C3A1705A4255Q39906454-091A1C3B-C375-4553-9BE4-F199C69BACC1Q39980778-AC58BD3F-4175-42E3-BE4A-50C8FAB3C54EQ40141217-28389DE2-9C70-4BA1-B973-42272F747B8FQ42418341-15BF7535-4B58-469C-AB75-65FB37A26BD1Q43212996-5DC07DDF-2571-47FD-AADF-AD818A207303
P2860
The second loop of occludin is required for suppression of Raf1-induced tumor growth.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
The second loop of occludin is required for suppression of Raf1-induced tumor growth.
@en
type
label
The second loop of occludin is required for suppression of Raf1-induced tumor growth.
@en
prefLabel
The second loop of occludin is required for suppression of Raf1-induced tumor growth.
@en
P2093
P2860
P356
P1433
P1476
The second loop of occludin is required for suppression of Raf1-induced tumor growth.
@en
P2093
Charles A Parkos
Kenneth J Mandell
Randall J Mrsny
P2860
P2888
P304
P356
10.1038/SJ.ONC.1208634
P407
P50
P577
2005-06-01T00:00:00Z
P5875
P6179
1038873394