Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).

Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).

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http://www.wikidata.org/entity/Q40459248
Traumatic Acid Reduces Oxidative Stress and Enhances Collagen Biosynthesis in Cultured Human Skin FibroblastsLeukocyte proteinases in wound healing: roles in physiologic and pathologic processes.Bacterial lipopolysaccharide and tumor necrosis factor alpha synergistically increase expression of human endothelial adhesion molecules through activation of NF-kappaB and p38 mitogen-activated protein kinase signaling pathways.The oxidant-scavenging abilities in the oral cavity may be regulated by a collaboration among antioxidants in saliva, microorganisms, blood cells and polyphenols: a chemiluminescence-based studyLoss of Interleukin 1 Receptor Antagonist Enhances Susceptibility to Ebola Virus Infection.Are cationic antimicrobial peptides also 'double-edged swords'?Circulating histones are major mediators of systemic inflammation and cellular injury in patients with acute liver failure.When two is better than one: macrophages and neutrophils work in concert in innate immunity as complementary and cooperative partners of a myeloid phagocyte system.Nuclear histones: major virulence factors or just additional early sepsis markers? A comment.The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.From amino acids polymers, antimicrobial peptides, and histones, to their possible role in the pathogenesis of septic shock: a historical perspective.Amelioration of TNBS-induced colon inflammation in rats by phospholipase A2 inhibitor.TNF-α and LPS activate angiogenesis via VEGF and SIRT1 signalling in human dental pulp cells.Are histones real pathogenic agents in sepsis?Hemolysis of human red blood cells induced by the combination of diethyldithiocarbamate (DDC) and divalent metals: modulation by anaerobiosis, certain antioxidants and oxidants.Serum histones as biomarkers of the severity of heatstroke in dogs.Uric acid disrupts hypochlorous acid production and the bactericidal activity of HL-60 cells.
P2860

Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).

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http://www.wikidata.org/entity/Q40459248
description
1995 nî lūn-bûn
@nan
1995年の論文
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1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Cell damage in inflammatory an ...... s and cytokines (an overview).
@en
type
label
Cell damage in inflammatory an ...... s and cytokines (an overview).
@en
prefLabel
Cell damage in inflammatory an ...... s and cytokines (an overview).
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921
P356
P1433
P1476
Cell damage in inflammatory an ...... s and cytokines (an overview).
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921