Tumor necrosis factor-alpha induces transforming growth factor-beta1 expression in lung fibroblasts through the extracellular signal-regulated kinase pathway. - Wikidata - awgldk - TriplyDB

Tumor necrosis factor-alpha induces transforming growth factor-beta1 expression in lung fibroblasts through the extracellular signal-regulated kinase pathway.

Tumor necrosis factor-alpha induces transforming growth factor-beta1 expression in lung fibroblasts through the extracellular signal-regulated kinase pathway.

http://www.wikidata.org/entity/Q40470243

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alpha2-antiplasmin is associated with the progression of fibrosis Mesenchymal stem cells produce Wnt isoforms and TGF-beta1 that mediate proliferation and procollagen expression by lung fibroblasts Immunotherapy of systemic sclerosis Myofibroblasts and lung fibrosis induced by carbon nanotube exposure Targeting the MEK1 cascade in lung epithelium inhibits proliferation and fibrogenesis by asbestos Pulmonary endpoints (lung carcinomas and asbestosis) following inhalation exposure to asbestos Atomic layer deposition coating of carbon nanotubes with aluminum oxide alters pro-fibrogenic cytokine expression by human mononuclear phagocytes in vitro and reduces lung fibrosis in mice in vivo Mechanisms of action of inhaled fibers, particles and nanoparticles in lung and cardiovascular diseases Multiwalled Carbon Nanotube Functionalization with High Molecular Weight Hyaluronan Significantly Reduces Pulmonary Injury Novel pathway compendium analysis elucidates mechanism of pro-angiogenic synthetic small molecule Non-infectious pulmonary complications of newer biological agents for rheumatic diseases--a systematic literature review.Laser capture microdissection reveals dose-response of gene expression in situ consequent to asbestos exposure.Basics of Radiation Biology When Treating Hyperproliferative Benign Diseases.TNF-alpha induces TGF-beta1 expression in lung fibroblasts at the transcriptional level via AP-1 activation.The effect of the pro-inflammatory cytokine tumor necrosis factor-alpha on human joint capsule myofibroblasts.Pathophysiology of fistula formation in Crohn's disease.Nitric oxide activation of Erk1/2 regulates the stability and translation of mRNA transcripts containing CU-rich elements Small interfering RNAs (siRNAs) targeting TGF-beta1 mRNA suppress asbestos-induced expression of TGF-beta1 and CTGF in fibroblasts.Profibrotic phenotype of conjunctival fibroblasts from mucous membrane pemphigoid.Increased and correlated expression of connective tissue growth factor and transforming growth factor beta 1 in surgically removed periodontal tissues with chronic periodontitis Cytokine/anti-cytokine therapy - novel treatments for asthma?The role for dickkopf-homolog-1 in the pathogenesis of Crohn's disease-associated fistulae Increased expression of p21WAF1/CIP1 in kidney proximal tubules mediates fibrosis.Anti-Inflammatory Potential of Ethanolic Leaf Extract of Eupatorium adenophorum Spreng. Through Alteration in Production of TNF-α, ROS and Expression of Certain Genes Activation of IKK/NF-κB provokes renal inflammatory responses in guanylyl cyclase/natriuretic peptide receptor-A gene-knockout mice.Beneficial Effects of Qili Qiangxin Capsule on Lung Structural Remodeling in Ischemic Heart Failure via TGF-β1/Smad3 Pathway.Role of signal transducer and activator of transcription 1 in murine allergen-induced airway remodeling and exacerbation by carbon nanotubes.TGF beta 1 and TNF alpha potentiate nitric oxide production in astrocyte cultures by recruiting distinct subpopulations of cells to express NOS-2.Unraveling the signaling pathways promoting fibrosis in Dupuytren's disease reveals TNF as a therapeutic target Perforating folliculitis associated with tumour necrosis factor-alpha inhibitors administered for rheumatoid arthritis.Vitamin D regulation of OX40 ligand in immune responses to Aspergillus fumigatus.Endostatin, an angiogenesis inhibitor, ameliorates bleomycin-induced pulmonary fibrosis in rats.Targeting TNF-alpha: a novel therapeutic approach for asthma Peptide-mediated inhibition of mitogen-activated protein kinase-activated protein kinase-2 ameliorates bleomycin-induced pulmonary fibrosis Cigarette smoke, inflammation, and lung injury: a mechanistic perspective.High-fat diet induces lung remodeling in ApoE-deficient mice: an association with an increase in circulatory and lung inflammatory factors.Use of biologicals as immunotherapy in asthma and related diseases.Portrait of inflammatory response to ionizing radiation treatment.A mechanistic review of silica-induced inhalation toxicity.TNF-α-induced NF-κB activation promotes myofibroblast differentiation of LR-MSCs and exacerbates bleomycin-induced pulmonary fibrosis.

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P2860

Tumor necrosis factor-alpha induces transforming growth factor-beta1 expression in lung fibroblasts through the extracellular signal-regulated kinase pathway.

http://www.wikidata.org/entity/Q40470243

description

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American Journal of Respiratory Cell and Molecular Biology

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Arnold R Brody

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Deborah E Sullivan

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Derek Pociask

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MaryBeth Ferris

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P2860

Growth factors in idiopathic pulmonary fibrosis: relative roles

Increased phosphorylated extracellular signal-regulated kinase immunoreactivity associated with proliferative and morphologic lung alterations after chrysotile asbestos inhalation in mice

Specificity and mechanism of action of some commonly used protein kinase inhibitors

Reduced tumor necrosis factor-alpha and transforming growth factor-beta1 expression in the lungs of inbred mice that fail to develop fibroproliferative lesions consequent to asbestos exposure

Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases

Matrix metalloproteinase-9 contributes to choroidal neovascularization.

Up-regulation of nucleolin mRNA and protein in peripheral blood mononuclear cells by extracellular-regulated kinase.

Quantitative reverse transcription-polymerase chain reaction to study mRNA decay: comparison of endpoint and real-time methods.

Regulation of mRNA stability in mammalian cells.

Extracellular signal-regulated kinases 1 and 2 participate in interleukin-17 plus tumor necrosis factor-alpha-induced stabilization of interleukin-6 mRNA in human pancreatic myofibroblasts.

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32

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15653932

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