Developmental lag in superoxide dismutases relative to other antioxidant enzymes in premyelinated human telencephalic white matter. - Wikidata - awgldk - TriplyDB

Developmental lag in superoxide dismutases relative to other antioxidant enzymes in premyelinated human telencephalic white matter.

Developmental lag in superoxide dismutases relative to other antioxidant enzymes in premyelinated human telencephalic white matter.

http://www.wikidata.org/entity/Q40479376

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Increased inflammatory markers are associated with early periventricular leukomalacia Delayed myelination in an intrauterine growth retardation model is mediated by oxidative stress upregulating bone morphogenetic protein 4 Pathophysiology of glia in perinatal white matter injury.Perinatal white matter injury: the changing spectrum of pathology and emerging insights into pathogenetic mechanisms.The encephalopathy of prematurity--brain injury and impaired brain development inextricably intertwined.The diagnosis, management, and postnatal prevention of intraventricular hemorrhage in the preterm neonate.The encephalopathy of prematurity: one pediatric neuropathologist's perspective.Prenatal hypoxic-ischemic insult changes the distribution and number of NADPH-diaphorase cells in the cerebellum.Developmental regulation of alpha-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid receptor subunit expression in forebrain and relationship to regional susceptibility to hypoxic/ischemic injury. II. Human cerebral white matter and cortex.Neurologic Correlates of Gait Abnormalities in Cerebral Palsy: Implications for Treatment.The developing oligodendrocyte: key cellular target in brain injury in the premature infant Metabolic maturation of white matter is altered in preterm infants.Oxidative stress disrupts oligodendrocyte maturation.Animal models of periventricular leukomalacia.Systemic prenatal insults disrupt telencephalon development: implications for potential interventions.Brainstem deficiency of the 14-3-3 regulator of serotonin synthesis: a proteomics analysis in the sudden infant death syndrome Oxidative and nitrative injury in periventricular leukomalacia: a review.The role of inflammation in perinatal brain injury.White matter injury in the preterm infant: pathology and mechanisms.Oxidative stress in immature brain after traumatic brain injury.The potential role of mitochondria in pediatric traumatic brain injury.Oxidative stress and the antioxidant enzyme system in the developing brain Pathogenesis of cerebral white matter injury of prematurity.The effects of therapeutic hypothermia on cerebral metabolism in neonates with hypoxic-ischemic encephalopathy: An in vivo 1H-MR spectroscopy study Brain injury in premature infants: a complex amalgam of destructive and developmental disturbances.Developmental regulation of the NMDA receptor subunits, NR3A and NR1, in human prefrontal cortex.12/15-lipoxygenase expression is increased in oligodendrocytes and microglia of periventricular leukomalacia 17beta-estradiol protects against hypoxic/ischemic white matter damage in the neonatal rat brain.Neurobiology of injury to the developing brain.Oligodendrocyte pathophysiology and treatment strategies in cerebral ischemia.Neurodevelopmental outcome in congenital diaphragmatic hernia: Evaluation, predictors and outcome.Preclinical Models of Encephalopathy of Prematurity.Prenatal Systemic Hypoxia-Ischemia and Oligodendroglia Loss in Cerebellum.Therapeutic Strategies for Leukodystrophic Disorders Resulting from Perinatal Asphyxia: Focus on Myelinating Oligodendrocytes.Modeling the encephalopathy of prematurity in animals: the important role of translational research.Towards improved animal models of neonatal white matter injury associated with cerebral palsy.Developmental regulation of group I metabotropic glutamate receptors in the premature brain and their protective role in a rodent model of periventricular leukomalacia.Developmental stage of oligodendrocytes determines their response to activated microglia in vitro.Selective vulnerability of preterm white matter to oxidative damage defined by F2-isoprostanes.Improvement of hypoxia-ischemia-induced white matter injury in immature rat brain by ethyl pyruvate.

P2860

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P2860

Developmental lag in superoxide dismutases relative to other antioxidant enzymes in premyelinated human telencephalic white matter.

http://www.wikidata.org/entity/Q40479376

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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Developmental lag in superoxid ...... an telencephalic white matter.

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Developmental lag in superoxid ...... an telencephalic white matter.

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Developmental lag in superoxid ...... an telencephalic white matter.

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Developmental lag in superoxid ...... an telencephalic white matter.

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P2093

Felicia Trachtenberg

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Hannah C Kinney

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Joseph J Volpe

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Natalia S Borenstein

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Paul A Rosenberg

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Rebecca D Folkerth

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Richard A Belliveau

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Robin L Haynes

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P2860

Superoxide activates mitochondrial uncoupling proteins

Developmental changes in murine brain antioxidant enzymes

Changes of hippocampal Cu/Zn-superoxide dismutase after kainate treatment in the rat

The mitochondrial generation of hydrogen peroxide. General properties and effect of hyperbaric oxygen

Perinatal infection, fetal inflammatory response, white matter damage, and cognitive limitations in children born preterm.

Perinatal brain injury: from pathogenesis to neuroprotection.

Intrauterine infection and prematurity.

Models of white matter injury: comparison of infectious, hypoxic-ischemic, and excitotoxic insults.

Control of eye orientation: where does the brain's role end and the muscle's begin?

Inhibition of phosphatidylinositol 3-kinase induces nitric-oxide synthase in lipopolysaccharide- or cytokine-stimulated C6 glial cells.

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