G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

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Instructive role of M-CSF on commitment of bipotent myeloid cells involves ERK-dependent positive and negative signaling.Transcription factor Fli-1 expression by bone marrow cells in chronic myeloproliferative disorders is independent of an underlying JAK2 (V617F) mutation.

P2860

Q38838269-B8FC4FFA-DC76-4258-8844-E6B397C94B4C Q50927508-F4E25206-1CC4-4988-B183-DD43E40388E1

P2860

G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

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http://www.wikidata.org/entity/Q40489436

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2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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@zh-sg

2004年學術文章

@yue

2004年學術文章

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2004年學術文章

@zh-hant

name

G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

@en

type

Item

label

G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

@en

prefLabel

G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

@en

P1476

G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

@en

P2093

Jolyn Wei

http://www.w3.org/2001/XMLSchema#string

Kathleen M Sakamoto

http://www.w3.org/2001/XMLSchema#string

Patricia Mora-Garcia

http://www.w3.org/2001/XMLSchema#string

P2860

Hemorrhage, impaired hematopoiesis, and lethality in mouse embryos carrying a targeted disruption of the Fli1 transcription factor

Identification and mapping of a common proviral integration site Fli-1 in erythroleukemia cells induced by Friend murine leukemia virus

Serine/threonine phosphorylation in cytokine signal transduction.

FLI-1 inhibits differentiation and induces proliferation of primary erythroblasts.

Clinical applications of hematopoietic growth factors in pediatric oncology.

Characterization of messenger ribonucleoprotein and messenger RNA from KB cells.

Transcriptional regulators and myelopoiesis: the role of serum response factor and CREB as targets of cytokine signaling.

Immunotherapy in the prophylaxis and treatment of neonatal sepsis.

An immunological renal disease in transgenic mice that overexpress Fli-1, a member of the ets family of transcription factor genes.

Granulocyte-macrophage colony-stimulating factor and interleukin-3 signaling pathways converge on the CREB-binding site in the human egr-1 promoter.

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01.pdr.0000147729.55592.2c

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63-66

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scholarly article

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10.1203/01.PDR.0000147729.55592.2C

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English

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2004-11-19T00:00:00Z

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8170430

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P6179

1039498210

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P698

15557108

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G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

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P2860

P2860

G-CSF induces stabilization of ETS protein Fli-1 during myeloid cell development.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P577

P5875

P6179

P698

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P577

P5875

P6179

P698