Mutant copper-zinc superoxide dismutase binds to and destabilizes human low molecular weight neurofilament mRNA.
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Insulin-like growth factor-I for the treatment of amyotrophic lateral sclerosisStructural Characterization of Zinc-deficient Human Superoxide Dismutase and Implications for ALSThe complex molecular biology of amyotrophic lateral sclerosis (ALS)Analysis of novel NEFL mRNA targeting microRNAs in amyotrophic lateral sclerosisDNA-triggered aggregation of copper, zinc superoxide dismutase in the presence of ascorbate.Vascular endothelial growth factor prevents G93A-SOD1-induced motor neuron degeneration.The emerging role of guanine nucleotide exchange factors in ALS and other neurodegenerative diseases.Modeling ALS with iPSCs reveals that mutant SOD1 misregulates neurofilament balance in motor neurons.Glial nuclear aggregates of superoxide dismutase-1 are regularly present in patients with amyotrophic lateral sclerosis.Heat shock factor 1 over-expression protects against exposure of hydrophobic residues on mutant SOD1 and early mortality in a mouse model of amyotrophic lateral sclerosis.Neurofilament proteins in axonal regeneration and neurodegenerative diseases.Real-Time PCR-Coupled CE-SELEX for DNA Aptamer SelectionFrom Transcriptome to Noncoding RNAs: Implications in ALS Mechanism.Amyotrophic lateral sclerosis associated with mutations in the CuZn superoxide dismutase gene.Role of zinc in ALS.Altered microRNA expression profile in Amyotrophic Lateral Sclerosis: a role in the regulation of NFL mRNA levels.Mutant copper-zinc superoxide dismutase associated with amyotrophic lateral sclerosis binds to adenine/uridine-rich stability elements in the vascular endothelial growth factor 3'-untranslated region.RNA processing pathways in amyotrophic lateral sclerosis.The role of RNA processing in the pathogenesis of motor neuron degeneration.TDP-43 and FUS/TLS: sending a complex message about messenger RNA in amyotrophic lateral sclerosis?Polyanion binding accelerates the formation of stable and low-toxic aggregates of ALS-linked SOD1 mutant A4V.Human low molecular weight neurofilament (NFL) mRNA interacts with a predicted p190RhoGEF homologue (RGNEF) in humans.The roles of intrinsic disorder-based liquid-liquid phase transitions in the "Dr. Jekyll-Mr. Hyde" behavior of proteins involved in amyotrophic lateral sclerosis and frontotemporal lobar degeneration.Detection of a novel frameshift mutation and regions with homozygosis within ARHGEF28 gene in familial amyotrophic lateral sclerosis.Divalent-metal-dependent nucleolytic activity of Cu, Zn superoxide dismutase.TDP-43 in neurodegenerative disordersCytoskeletal changes in diseases of the nervous system
P2860
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P2860
Mutant copper-zinc superoxide dismutase binds to and destabilizes human low molecular weight neurofilament mRNA.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Mutant copper-zinc superoxide ...... lar weight neurofilament mRNA.
@en
type
label
Mutant copper-zinc superoxide ...... lar weight neurofilament mRNA.
@en
prefLabel
Mutant copper-zinc superoxide ...... lar weight neurofilament mRNA.
@en
P2093
P2860
P356
P1476
Mutant copper-zinc superoxide ...... lar weight neurofilament mRNA.
@en
P2093
Cheryl Leystra-Lantz
Michael J Strong
Wei-Wen Ge
Weiyan Wen
Wendy Strong
P2860
P304
P356
10.1074/JBC.M405065200
P407
P577
2004-10-25T00:00:00Z