Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms. - Wikidata - awgldk - TriplyDB

Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms.

Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms.

http://www.wikidata.org/entity/Q40522016

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HMGB1: endogenous danger signaling Graft rejection - endogenous or allogeneic?A cardiovascular drug rescues mice from lethal sepsis by selectively attenuating a late-acting proinflammatory mediator, high mobility group box 1 Galectin-9 prolongs the survival of septic mice by expanding Tim-3-expressing natural killer T cells and PDCA-1+ CD11c+ macrophages A hepatic protein, fetuin-A, occupies a protective role in lethal systemic inflammation An Agent-Based Model of a Hepatic Inflammatory Response to Salmonella: A Computational Study under a Large Set of Experimental Data Sepsis: in search of cure.A major ingredient of green tea rescues mice from lethal sepsis partly by inhibiting HMGB1 Sublethal doses of anthrax lethal toxin on the suppression of macrophage phagocytosis.High mobility group box 1 protein as a potential drug target for infection- and injury-elicited inflammation.Inflammatory response and immune regulation of high mobility group box-1 protein in treatment of sepsis.High-mobility group box 1 represents a potential marker of disease activity and novel therapeutic target in systemic lupus erythematosus.More tea for septic patients?--Green tea may reduce endotoxin-induced release of high mobility group box 1 and other pro-inflammatory cytokines.HMGB1 in health and disease.Metformin inhibits HMGB1 release in LPS-treated RAW 264.7 cells and increases survival rate of endotoxaemic mice EGCG stimulates autophagy and reduces cytoplasmic HMGB1 levels in endotoxin-stimulated macrophages Calcium/calmodulin-dependent protein kinase (CaMK) Ialpha mediates the macrophage inflammatory response to sepsis Apoptotic response through a high mobility box 1 protein-dependent mechanism in LPS/GalN-induced mouse liver failure and glycyrrhizin-mediated inhibition.The compromise of macrophage functions by hyperoxia is attenuated by ethacrynic acid via inhibition of NF-κB-mediated release of high-mobility group box-1.Receptor for advanced glycation end products - membrane type1 matrix metalloproteinase axis regulates tissue factor expression via RhoA and Rac1 activation in high-mobility group box-1 stimulated endothelial cells.The aqueous extract of a popular herbal nutrient supplement, Angelica sinensis, protects mice against lethal endotoxemia and sepsis.Role of HMGB1 in cardiovascular diseases Potential role of high mobility group box 1 in viral infectious diseases Hydrogen peroxide stimulates macrophages and monocytes to actively release HMGB1 Pruni cortex ameliorates skin inflammation possibly through HMGB1-NFκB pathway in house dust mite induced atopic dermatitis NC/Nga transgenic mice HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload Damage-associated molecular patterns (DAMPs) in preterm labor with intact membranes and preterm PROM: a study of the alarmin HMGB1 EGCG induces G-CSF expression and neutrophilia in experimental sepsis It Is Not Just Folklore: The Aqueous Extract of Mung Bean Coat Is Protective against Sepsis The in Vitro Immune-Modulating Properties of a Sweat Gland-Derived Antimicrobial Peptide Dermcidin.Endogenous control of immunity against infection: tenascin-C regulates TLR4-mediated inflammation via microRNA-155 Treatment of severe sepsis: where next? Current and future treatment approaches after the introduction of drotrecogin alfa.Induction of arthritis by high mobility group box chromosomal protein 1 is independent of tumour necrosis factor signalling.High-mobility group box 1 is associated with neurological outcome in patients with post-cardiac arrest syndrome after out-of-hospital cardiac arrest Bench-to-bedside review: High-mobility group box 1 and critical illness.Carbenoxolone blocks endotoxin-induced protein kinase R (PKR) activation and high mobility group box 1 (HMGB1) release.Relationship between vascular reactivity and expression of HMGB1 in a rat model of septic aorta.Thrombin inhibits HMGB1-mediated proinflammatory signaling responses when endothelial protein C receptor is occupied by its natural ligand.Caging a Beast in the Inflammation Arena: Use of Chinese Medicinal Herbs to Inhibit a Late Mediator of Lethal Sepsis, HMGB1.Annexin A5 increases survival in murine sepsis model by inhibiting HMGB1-mediated pro-inflammation and coagulation.

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Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms.

http://www.wikidata.org/entity/Q40522016

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Bacterial endotoxin stimulates ...... and TNF-dependent mechanisms.

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Bacterial endotoxin stimulates ...... and TNF-dependent mechanisms.

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Bacterial endotoxin stimulates ...... and TNF-dependent mechanisms.

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Journal of Leukocyte Biology

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Bacterial endotoxin stimulates ...... and TNF-dependent mechanisms.

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P2093

Andrew E Sama

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Beatriz Rendon-Mitchell

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Guoqian Chen

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Huan Yang

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Jianhua Li

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Mahendar Ochani

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Ping Wang

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Saijun Fan

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Xiaoling Qiang

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Lipopolysaccharide-binding protein mediates CD14-independent intercalation of lipopolysaccharide into phospholipid membranes

A protein kinase involved in the regulation of inflammatory cytokine biosynthesis

Resistance to endotoxin shock and reduced dissemination of gram-negative bacteria in CD14-deficient mice

The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes

CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion.

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10.1189/JLB.0404242

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5

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76

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Kevin J. Tracey

Srinivas M. Susarla

Sanna M. Goyert

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2004-08-26T00:00:00Z

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8379997

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15331624

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