Lipopolysaccharide-induced febrile convulsions in the rat: short-term sequelae.
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A swell in the armamentarium of antiepileptic drug targetsDeficient adolescent social behavior following early-life inflammation is ameliorated by augmentation of anandamide signaling.Fever, febrile seizures and epilepsy.Lipopolysaccharide potentiates hyperthermia-induced seizures.Rapid, Coordinate Inflammatory Responses after Experimental Febrile Status Epilepticus: Implications for Epileptogenesis.Sex-dependent effects of neonatal inflammation on adult inflammatory markers and behaviorViral-like brain inflammation during development causes increased seizure susceptibility in adult rats.Cytokines and brain excitability.Postnatal inflammation increases seizure susceptibility in adult rats.Cardiorespiratory control and cytokine profile in response to heat stress, hypoxia, and lipopolysaccharide (LPS) exposure during early neonatal period.Lack of association between TNF-α gene polymorphisms at position -308 A, -850T and risk of simple febrile convulsion in pediatric patients.Febrile seizures: mechanisms and relationship to epilepsyThe role of interleukin-1beta in febrile seizures.Origins of temporal lobe epilepsy: febrile seizures and febrile status epilepticus.Molecular cascades that mediate the influence of inflammation on epilepsy.The role of inflammation in epileptogenesis.Inflammation and epilepsy in the developing brain: clinical and experimental evidence.Acute Neuroinflammation Promotes Cell Responses to 1800 MHz GSM Electromagnetic Fields in the Rat Cerebral Cortex.Generation of Febrile Seizures and Subsequent Epileptogenesis.Febrile convulsions induced by the combination of lipopolysaccharide and low-dose kainic acid enhance seizure susceptibility, not epileptogenesis, in rats.Cytokines: a link between fever and seizures.Interactive effects of maternal cigarette smoke, heat stress, hypoxia, and lipopolysaccharide on neonatal cardiorespiratory and cytokine responses.Inflammation enhances epileptogenesis in the developing rat brainInflammation induced by LPS enhances epileptogenesis in immature rat and may be partially reversed by IL1RA.Atypical febrile seizures, mesial temporal lobe epilepsy, and dual pathology.The anti-seizure drugs vinpocetine and carbamazepine, but not valproic acid, reduce inflammatory IL-1β and TNF-α expression in rat hippocampus.Pharmacological Tools to Activate Microglia and their Possible use to Study Neural Network Patho-physiology.Postnatal programming of the innate immune response.Increased excitability and molecular changes in adult rats after a febrile seizure.Neuroinflammation imaging markers for epileptogenesis.Inflammation exacerbates seizure-induced injury in the immature brain.Causal links between brain cytokines and experimental febrile convulsions in the rat.Inflammation contributes to seizure-induced hippocampal injury in the neonatal rat brain.An enhanced home cage modulates hypothalamic CRH-ir labeling in juvenile rats, with and without sub-threshold febrile convulsions.Neonatal Seizure Models to Study Epileptogenesis.
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P2860
Lipopolysaccharide-induced febrile convulsions in the rat: short-term sequelae.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Lipopolysaccharide-induced febrile convulsions in the rat: short-term sequelae.
@en
type
label
Lipopolysaccharide-induced febrile convulsions in the rat: short-term sequelae.
@en
prefLabel
Lipopolysaccharide-induced febrile convulsions in the rat: short-term sequelae.
@en
P2860
P1433
P1476
Lipopolysaccharide-induced febrile convulsions in the rat: short-term sequelae
@en
P2093
James G Heida
Lysa Boissé
P2860
P304
P356
10.1111/J.0013-9580.2004.13704.X
P577
2004-11-01T00:00:00Z