The oral administration of retinoic acid enhances nociceptive withdrawal reflexes in rats with soft-tissue inflammation.
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All-trans retinoic acid induces COX-2 and prostaglandin E2 synthesis in SH-SY5Y human neuroblastoma cells: involvement of retinoic acid receptors and extracellular-regulated kinase 1/2Retinoids activate the irritant receptor TRPV1 and produce sensory hypersensitivityNitroparacetamol (NCX-701) and pain: first in a series of novel analgesics.LE135, a retinoid acid receptor antagonist, produces pain through direct activation of TRP channels.Vitamin A active metabolite, all-trans retinoic acid, induces spinal cord sensitization. II. Effects after intrathecal administration.Vitamin A active metabolite, all-trans retinoic acid, induces spinal cord sensitization. I. Effects after oral administration.Upregulation of cyclooxygenases by retinoic acid in rat mesangial cells.Kinase-dependent, retinoic acid receptor-independent up-regulation of cyclooxygenase-2 by all-trans retinoic acid in human mesangial cells.NCX 2057, a novel NO-releasing derivative of ferulic acid, suppresses inflammatory and nociceptive responses in in vitro and in vivo models.Anti-inflammatory effect of retinoic acid on prostaglandin synthesis in cultured cortical astrocytes.
P2860
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P2860
The oral administration of retinoic acid enhances nociceptive withdrawal reflexes in rats with soft-tissue inflammation.
description
2004 nî lūn-bûn
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2004年の論文
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2004年論文
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2004年論文
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name
The oral administration of ret ...... with soft-tissue inflammation.
@en
type
label
The oral administration of ret ...... with soft-tissue inflammation.
@en
prefLabel
The oral administration of ret ...... with soft-tissue inflammation.
@en
P2093
P1476
The oral administration of ret ...... with soft-tissue inflammation
@en
P2093
Moreno-Manzano V
Romero-Sandoval EA
P2888
P304
P356
10.1007/S00011-004-1261-5
P577
2004-06-25T00:00:00Z