Susceptibility to SARS coronavirus S protein-driven infection correlates with expression of angiotensin converting enzyme 2 and infection can be blocked by soluble receptor.
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SARS molecular epidemiology: a Chinese fairy tale of controlling an emerging zoonotic disease in the genomics era.DESC1 and MSPL activate influenza A viruses and emerging coronaviruses for host cell entry.Differential downregulation of ACE2 by the spike proteins of severe acute respiratory syndrome coronavirus and human coronavirus NL63.Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.Antiviral drugs specific for coronaviruses in preclinical developmentNovel inhibitors of severe acute respiratory syndrome coronavirus entry that act by three distinct mechanismsEvidence that TMPRSS2 activates the severe acute respiratory syndrome coronavirus spike protein for membrane fusion and reduces viral control by the humoral immune response.Cleavage and activation of the severe acute respiratory syndrome coronavirus spike protein by human airway trypsin-like proteaseLongitudinally profiling neutralizing antibody response to SARS coronavirus with pseudotypes.Current status of anti-SARS agents.Potential antivirals and antiviral strategies against SARS coronavirus infections.Animal origins of the severe acute respiratory syndrome coronavirus: insight from ACE2-S-protein interactions.Modulation of TNF-alpha-converting enzyme by the spike protein of SARS-CoV and ACE2 induces TNF-alpha production and facilitates viral entryThe human/animal interface: emergence and resurgence of zoonotic infectious diseases.TMPRSS2 activates the human coronavirus 229E for cathepsin-independent host cell entry and is expressed in viral target cells in the respiratory epithelium.Different host cell proteases activate the SARS-coronavirus spike-protein for cell-cell and virus-cell fusion.Clathrin-dependent entry of severe acute respiratory syndrome coronavirus into target cells expressing ACE2 with the cytoplasmic tail deleted.Highly conserved regions within the spike proteins of human coronaviruses 229E and NL63 determine recognition of their respective cellular receptors.Identification and characterization of a Penaeus monodon lymphoid cell-expressed receptor for the yellow head virus.Receptor and viral determinants of SARS-coronavirus adaptation to human ACE2.Coronaviral hypothetical and structural proteins were found in the intestinal surface enterocytes and pneumocytes of severe acute respiratory syndrome (SARS).Selective and specific regulation of ectodomain shedding of angiotensin-converting enzyme 2 by tumor necrosis factor alpha-converting enzyme.A 12-year follow-up study of combined treatment of post-severe acute respiratory syndrome patients with femoral head necrosis.Severe acute respiratory syndromeAttenuation of replication by a 29 nucleotide deletion in SARS-coronavirus acquired during the early stages of human-to-human transmission
P2860
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P2860
Susceptibility to SARS coronavirus S protein-driven infection correlates with expression of angiotensin converting enzyme 2 and infection can be blocked by soluble receptor.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Susceptibility to SARS coronav ...... e blocked by soluble receptor.
@en
type
label
Susceptibility to SARS coronav ...... e blocked by soluble receptor.
@en
prefLabel
Susceptibility to SARS coronav ...... e blocked by soluble receptor.
@en
P2093
P1476
Susceptibility to SARS coronav ...... e blocked by soluble receptor.
@en
P2093
Georg H Fey
Heike Hofmann
Mandy Krumbiegel
Martina Geier
Matthias Peipp
Thomas Gramberg
P304
P356
10.1016/J.BBRC.2004.05.114
P577
2004-07-01T00:00:00Z