Energy dysfunction as a predictor of outcome after moderate or severe head injury: indices of oxygen, glucose, and lactate metabolism. - Wikidata - awgldk - TriplyDB

Energy dysfunction as a predictor of outcome after moderate or severe head injury: indices of oxygen, glucose, and lactate metabolism.

Energy dysfunction as a predictor of outcome after moderate or severe head injury: indices of oxygen, glucose, and lactate metabolism.

http://www.wikidata.org/entity/Q40558325

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Metabolic crisis in severely head-injured patients: is ischemia just the tip of the iceberg?Intensive Care Treatment in Traumatic Brain Injury Traumatic Brain Injury pathophysiology and treatments: early, intermediate, and late phases post-injury Cerebral metabolism following traumatic brain injury: new discoveries with implications for treatment Metabolic crisis without brain ischemia is common after traumatic brain injury: a combined microdialysis and positron emission tomography study Lactate shuttling and lactate use as fuel after traumatic brain injury: metabolic considerations Dynamic metabolic response to multiple spreading depolarizations in patients with acute brain injury: an online microdialysis study.Clinical review: neuromonitoring - an update.Glucose metabolism following human traumatic brain injury: methods of assessment and pathophysiological findings.Probing astrocyte metabolism in vivo: proton magnetic resonance spectroscopy in the injured and aging brain Role of Metabolomics in Traumatic Brain Injury Research.Structural imaging of mild traumatic brain injury may not be enough: overview of functional and metabolic imaging of mild traumatic brain injury.Evidence for the mitochondrial lactate oxidation complex in rat neurons: demonstration of an essential component of brain lactate shuttles Use of hyperbaric oxygen in traumatic brain injury: retrospective analysis of data of 20 patients treated at a tertiary care centre.Acute gonadotroph and somatotroph hormonal suppression after traumatic brain injury.Early metabolic crisis-related brain atrophy and cognition in traumatic brain injury.High blood glucose does not adversely affect outcome in moderately brain-injured rodents.Neuroimaging patterns associated with motor control in traumatic brain injury.Hyperbaric oxygen in traumatic brain injury.Glucose and oxygen metabolism after penetrating ballistic-like brain injury Mitochondrial response in a toddler-aged swine model following diffuse non-impact traumatic brain injury.Endogenous Nutritive Support after Traumatic Brain Injury: Peripheral Lactate Production for Glucose Supply via Gluconeogenesis Lactate: brain fuel in human traumatic brain injury: a comparison with normal healthy control subjects.Test-retest reproducibility of a rapid method to measure brain oxygen metabolism.Hyperpolarized 13C NMR observation of lactate kinetics in skeletal muscle.Mouse model of diffuse brain damage following anoxia, evaluated by a new assay of generalized arousal Brain temperature: physiology and pathophysiology after brain injury.Depression of whole-brain oxygen extraction fraction is associated with poor outcome in pediatric traumatic brain injury.Improvement of Neuroenergetics by Hypertonic Lactate Therapy in Patients with Traumatic Brain Injury Is Dependent on Baseline Cerebral Lactate/Pyruvate Ratio.Differential temporal profile of lowered blood glucose levels (3.5 to 6.5 mmol/l versus 5 to 8 mmol/l) in patients with severe traumatic brain injury.Pathophysiology of hypopituitarism in the setting of brain injury Variation in chronic nicotinamide treatment after traumatic brain injury can alter components of functional recovery independent of histological damage.Cerebral extracellular lactate increase is predominantly nonischemic in patients with severe traumatic brain injury Human Serum Metabolites Associate With Severity and Patient Outcomes in Traumatic Brain Injury Lactate uptake by the injured human brain: evidence from an arteriovenous gradient and cerebral microdialysis study.Vessel-specific quantification of blood oxygenation with T2-relaxation-under-phase-contrast MRI.A review of perioperative glucose control in the neurosurgical population PET in Cerebrovascular Disease.The pathophysiology of concussions in youth.Therapeutic hypothermia for traumatic brain injury.

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P2860

Energy dysfunction as a predictor of outcome after moderate or severe head injury: indices of oxygen, glucose, and lactate metabolism.

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P2860

Mitochondrial participation in ischemic and traumatic neural cell death

Random-effects models for longitudinal data

Longitudinal data analysis for discrete and continuous outcomes

Effects of hyperbaric oxygenation therapy on cerebral metabolism and intracranial pressure in severely brain injured patients.

Metabolic recovery following human traumatic brain injury based on FDG-PET: time course and relationship to neurological disability.

Assessment of outcome after severe brain damage.

Pyruvate in organ transplantation.

Changes in cortical extracellular levels of energy-related metabolites and amino acids following concussive brain injury in rats.

Cerebral circulation and metabolism after severe traumatic brain injury: the elusive role of ischemia.

Cerebral blood flow, arteriovenous oxygen difference, and outcome in head injured patients

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