Spacial and Temporal Patterns of Gene Expression After Cardiac MEK1 Gene Transfer Improve Post-Infarction Remodeling Without Inducing Global Hypertrophy.

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The Gene Therapy Resource Program: A Decade of Dedication to Translational Research by the National Heart, Lung, and Blood Institute.Atrial ERK1/2 activation in the embryo leads to incomplete Septal closure: a novel mouse model of atrial Septal defect.

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Spacial and Temporal Patterns of Gene Expression After Cardiac MEK1 Gene Transfer Improve Post-Infarction Remodeling Without Inducing Global Hypertrophy.

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Spacial and Temporal Patterns ...... t Inducing Global Hypertrophy.

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Che-Chung Yeh

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Michael J Mann

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Yanying Fan

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Yi-Lin Yang

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Heart Disease and Stroke Statistics--2013 Update: A Report From the American Heart Association

Gene therapy for cardiovascular disease: advances in vector development, targeting, and delivery for clinical translation

Cardiac AAV9-S100A1 gene therapy rescues post-ischemic heart failure in a preclinical large animal model

S100A1 genetically targeted therapy reverses dysfunction of human failing cardiomyocytes.

The primary structure of MEK, a protein kinase that phosphorylates the ERK gene product

PKC alpha regulates the hypertrophic growth of cardiomyocytes through extracellular signal-regulated kinase1/2 (ERK1/2)

A myocardium tropic adeno-associated virus (AAV) evolved by DNA shuffling and in vivo selection.

Adenovirus-mediated gene transfer to cerebral circulation.

Targeting phospholamban by gene transfer in human heart failure.

Highly efficient gene transfer into adult ventricular myocytes by recombinant adenovirus.

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10.1002/JCB.25743

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Spacial and Temporal Patterns of Gene Expression After Cardiac MEK1 Gene Transfer Improve Post-Infarction Remodeling Without Inducing Global Hypertrophy.

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P2860

P2860

Spacial and Temporal Patterns of Gene Expression After Cardiac MEK1 Gene Transfer Improve Post-Infarction Remodeling Without Inducing Global Hypertrophy.

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