Brucella abortus rough mutants are cytopathic for macrophages in culture.
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Entry and elimination of marine mammal Brucella spp. by hooded seal (Cystophora cristata) alveolar macrophages in vitroProteomic analysis of detergent resistant membrane domains during early interaction of macrophages with rough and smooth Brucella melitensisEntrance and survival of Brucella pinnipedialis hooded seal strain in human macrophages and epithelial cellsContrasting Lifestyles Within the Host CellMutations of Francisella novicida that alter the mechanism of its phagocytosis by murine macrophagesAdvancement of knowledge of Brucella over the past 50 years.Mariner mutagenesis of Brucella melitensis reveals genes with previously uncharacterized roles in virulence and survival.RNAi screen of endoplasmic reticulum-associated host factors reveals a role for IRE1alpha in supporting Brucella replication.Caspase-2 mediated apoptotic and necrotic murine macrophage cell death induced by rough Brucella abortusIn vitro viability and cytotoxicity testing and same-well multi-parametric combinations for high throughput screeningFrancisella tularensis Schu S4 O-antigen and capsule biosynthesis gene mutants induce early cell death in human macrophages.Regulation of the mitogen-activated protein kinases by Brucella spp. expressing a smooth and rough phenotype: relationship to pathogen invasiveness.Mechanism of Asp24 upregulation in Brucella abortus rough mutant with a disrupted O-antigen export system and effect of Asp24 in bacterial intracellular survival.Deep-sequencing analysis of the mouse transcriptome response to infection with Brucella melitensis strains of differing virulence.The AS87_04050 gene is involved in bacterial lipopolysaccharide biosynthesis and pathogenicity of Riemerella anatipestiferCaspase-2-dependent dendritic cell death, maturation, and priming of T cells in response to Brucella abortus infection.Brucella abortus rough mutants induce macrophage oncosis that requires bacterial protein synthesis and direct interaction with the macrophage.Evaluation of protection afforded by Brucella abortus and Brucella melitensis unmarked deletion mutants exhibiting different rates of clearance in BALB/c mice.Microarray-based identification of differentially expressed genes in intracellular Brucella abortus within RAW264.7 cellsLipopolysaccharide-Deficient Brucella Variants Arise Spontaneously during Infection.Quorum-sensing and BvrR/BvrS regulation, the type IV secretion system, cyclic glucans, and BacA in the virulence of Brucella ovis: similarities to and differences from smooth brucellaeExperimental Challenge of Atlantic Cod (Gadus morhua) with a Brucella pinnipedialis Strain from Hooded Seal (Cystophora cristata)Ontology-based representation and analysis of host-Brucella interactions.Characterization of recombinant B. abortus strain RB51SOD toward understanding the uncorrelated innate and adaptive immune responses induced by RB51SOD compared to its parent vaccine strain RB51Toll-like receptors are critical for clearance of Brucella and play different roles in development of adaptive immunity following aerosol challenge in miceImmunization with a single dose of a microencapsulated Brucella melitensis mutant enhances protection against wild-type challenge.Brucellosis vaccines: assessment of Brucella melitensis lipopolysaccharide rough mutants defective in core and O-polysaccharide synthesis and exportGenomic island 2 of Brucella melitensis is a major virulence determinant: functional analyses of genomic islands.The Brucella abortus S19 DeltavjbR live vaccine candidate is safer than S19 and confers protection against wild-type challenge in BALB/c mice when delivered in a sustained-release vehicleSurvival of the fittest: how Brucella strains adapt to their intracellular niche in the host.Internal affairs: investigating the Brucella intracellular lifestyle.Analyses of Brucella pathogenesis, host immunity, and vaccine targets using systems biology and bioinformatics.Brucella melitensis triggers time-dependent modulation of apoptosis and down-regulation of mitochondrion-associated gene expression in mouse macrophages.Transcriptional regulator GntR of Brucella abortus regulates cytotoxicity, induces the secretion of inflammatory cytokines and affects expression of the type IV secretion system and quorum sensing system in macrophages.The Brucella melitensis M5-90 phosphoglucomutase (PGM) mutant is attenuated and confers protection against wild-type challenge in BALB/c mice.TceSR two-component regulatory system of Brucella melitensis 16M is involved in invasion, intracellular survival and regulated cytotoxicity for macrophages.Brucella dissociation is essential for macrophage egress and bacterial dissemination.Evidence of Brucella abortus OPS dictating uptake and restricting NF-kappaB activation in murine macrophages.Cytotoxicity in macrophages infected with rough Brucella mutants is type IV secretion system dependent.Systems Biology Analysis of Temporal In vivo Brucella melitensis and Bovine Transcriptomes Predicts host:Pathogen Protein-Protein Interactions.
P2860
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P2860
Brucella abortus rough mutants are cytopathic for macrophages in culture.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Brucella abortus rough mutants are cytopathic for macrophages in culture.
@en
type
label
Brucella abortus rough mutants are cytopathic for macrophages in culture.
@en
prefLabel
Brucella abortus rough mutants are cytopathic for macrophages in culture.
@en
P2860
P1476
Brucella abortus rough mutants are cytopathic for macrophages in culture.
@en
P2093
Jianwu Pei
Thomas A Ficht
P2860
P304
P356
10.1128/IAI.72.1.440-450.2004
P407
P577
2004-01-01T00:00:00Z