MKP-1-induced dephosphorylation of extracellular signal-regulated kinase is essential for triggering nitric oxide-induced apoptosis in human breast cancer cell lines: implications in breast cancer.
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Specific activation of the glucocorticoid receptor and modulation of signal transduction pathways in human lens epithelial cellsExtracellular signal-regulated kinase 1/2 activation, via downregulation of mitogen-activated protein kinase phosphatase 1, mediates sex differences in desoxycorticosterone acetate-salt hypertension vascular reactivity.Role of soluble guanylyl cyclase-cyclic GMP signaling in tumor cell proliferation.Nitric oxide regulates angiogenesis through a functional switch involving thrombospondin-1.Down-regulation of vitamin D receptor in mammospheres: implications for vitamin D resistance in breast cancer and potential for combination therapy.Hypoxic inducible factor 1alpha, extracellular signal-regulated kinase, and p53 are regulated by distinct threshold concentrations of nitric oxide.Induction of a feed forward pro-apoptotic mechanistic loop by nitric oxide in a human breast cancer modelOxidative stress and ERK1/2 phosphorylation as predictors of outcome in hepatocellular carcinoma patients treated with sorafenib plus octreotide LAR.Molecular targets and oxidative stress biomarkers in hepatocellular carcinoma: an overview.NO to breast: when, why and why not?Nitric oxide release: part III. Measurement and reporting.Increased sensitivity of African American triple negative breast cancer cells to nitric oxide-induced mitochondria-mediated apoptosis.Vanadate prevents glucocorticoid-induced apoptosis of osteoblasts in vitro and osteocytes in vivo.Increased Expression of Beige/Brown Adipose Markers from Host and Breast Cancer Cells Influence Xenograft Formation in Mice.Oxidative stress specifically downregulates survivin to promote breast tumour formationInflammatory levels of nitric oxide inhibit airway epithelial cell migration by inhibition of the kinase ERK1/2 and activation of hypoxia-inducible factor-1 alpha.The chemical biology of nitric oxide: implications in cellular signalingMolecular mechanisms for discrete nitric oxide levels in cancer.Staphylococcal entertotoxins of the enterotoxin gene cluster (egcSEs) induce nitrous oxide- and cytokine dependent tumor cell apoptosis in a broad panel of human tumor cells.Cancer cell metabolism and the modulating effects of nitric oxide.Dual specificity phosphatase 1 expression inversely correlates with NF-κB activity and expression in prostate cancer and promotes apoptosis through a p38 MAPK dependent mechanism.Nitric oxide is a mediator of antiproliferative effects induced by proinflammatory cytokines on pancreatic beta cells.
P2860
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P2860
MKP-1-induced dephosphorylation of extracellular signal-regulated kinase is essential for triggering nitric oxide-induced apoptosis in human breast cancer cell lines: implications in breast cancer.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
MKP-1-induced dephosphorylatio ...... implications in breast cancer.
@en
type
label
MKP-1-induced dephosphorylatio ...... implications in breast cancer.
@en
prefLabel
MKP-1-induced dephosphorylatio ...... implications in breast cancer.
@en
P2093
P1433
P1476
MKP-1-induced dephosphorylatio ...... implications in breast cancer
@en
P2093
Gautam Chaudhuri
Rajan Singh
Shehla Pervin
William A Freije
P304
P407
P577
2003-12-01T00:00:00Z