Hyperlipidemia in APOE2 transgenic mice is ameliorated by a truncated apoE variant lacking the C-terminal domain.
about
Apolipoprotein E - A Multifunctional Protein with Implications in Various Pathologies as a Result of Its Structural FeaturesLDL receptor deficiency or apoE mutations prevent remnant clearance and induce hypertriglyceridemia in mice.Generation of a recombinant apolipoprotein E variant with improved biological functions: hydrophobic residues (LEU-261, TRP-264, PHE-265, LEU-268, VAL-269) of apoE can account for the apoE-induced hypertriglyceridemia.
P2860
Hyperlipidemia in APOE2 transgenic mice is ameliorated by a truncated apoE variant lacking the C-terminal domain.
description
2002 nî lūn-bûn
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2002年の論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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2002年论文
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2002年论文
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2002年论文
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name
Hyperlipidemia in APOE2 transg ...... lacking the C-terminal domain.
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type
label
Hyperlipidemia in APOE2 transg ...... lacking the C-terminal domain.
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prefLabel
Hyperlipidemia in APOE2 transg ...... lacking the C-terminal domain.
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P2093
P2860
P50
P1476
Hyperlipidemia in APOE2 transg ...... lacking the C-terminal domain
@en
P2093
André van der Zee
Gery Gerritsen
Ko Willems van Dijk
Louis M Havekes
P2860
P304
P356
10.1194/JLR.M200313-JLR200
P577
2002-10-16T00:00:00Z