Robustness and power of the unified model in the analysis of quantitative measurements.
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Sources of interindividual variation in the quantitative levels of apolipoprotein B in pedigrees ascertained through a lipid clinicSegregation analysis of apolipoprotein A1 levels in families of adolescents: a community-based study in Taiwan.Heritability and major gene effects on left ventricular mass in the Chinese population: a family study.Identification of QTL with effects on intramuscular fat content and fatty acid composition in a Duroc x Large White crossA common genetic mechanism determines plasma apolipoprotein B levels and dense LDL subfraction distribution in familial combined hyperlipidemia.Heritability of cellular radiosensitivity: a marker of low-penetrance predisposition genes in breast cancer?Segregation analysis of fat mass and other body composition measures derived from underwater weighing.Influence of genotype-dependent effects of covariates on the outcome of segregation analysis of the body mass index.Segregation analysis detects a major gene controlling blood infection levels in human malaria.Evidence, from combined segregation and linkage analysis, that a variant of the angiotensin I-converting enzyme (ACE) gene controls plasma ACE levels.Evidence for the segregation of a major gene in human susceptibility/resistance to infection by Schistosoma mansoni.Segregation analysis of low levels of high-density lipoprotein cholesterol in the collaborative Lipid Research Clinics Program Family Study.The genetic and environmental sources of body mass index variability: the Muscatine Ponderosity Family Study.Detection of major genes for susceptibility to leprosy and its subtypes in a Caribbean island: Desirade island.Genetic determination of plasma apolipoprotein AI in a population-based sample.Evidence for a major gene controlling susceptibility to tegumentary leishmaniasis in a recently exposed Bolivian population.Segregation analysis of non-insulin-dependent diabetes mellitus in Pima Indians: evidence for a major-gene effect.Segregation and linkage analysis of serum angiotensin I-converting enzyme levels: evidence for two quantitative-trait loci.Genetic and environmental sources of fibrinogen variability in Israeli families: the Kibbutzim Family Study.A major gene effect controls resistance to cariesDetection of a major gene for heterocellular hereditary persistence of fetal hemoglobin after accounting for genetic modifiers.Combined segregation and linkage analysis of Graves disease with a thyroid autoantibody diathesis.Conclusions of segregation analysis for family data generated under two-locus models.Testing separate families of segregation hypotheses: bootstrap methodsInferring a major gene for quantitative traits by using segregation analysis with tests on transmission probabilities: how often do we miss?POINTER correctly estimates the transmission probabilities under the general transmission model in the case of incomplete selection.The transmission probability model is useful to prevent false inference.
P2860
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P2860
Robustness and power of the unified model in the analysis of quantitative measurements.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
1986年论文
@zh
1986年论文
@zh-cn
name
Robustness and power of the unified model in the analysis of quantitative measurements.
@en
type
label
Robustness and power of the unified model in the analysis of quantitative measurements.
@en
prefLabel
Robustness and power of the unified model in the analysis of quantitative measurements.
@en
P2860
P1476
Robustness and power of the unified model in the analysis of quantitative measurements.
@en
P2093
Demenais F
P2860
P304
P407
P577
1986-02-01T00:00:00Z