Respiratory syncytial virus up-regulates TLR4 and sensitizes airway epithelial cells to endotoxin. - Wikidata - awgldk - TriplyDB

Respiratory syncytial virus up-regulates TLR4 and sensitizes airway epithelial cells to endotoxin.

Respiratory syncytial virus up-regulates TLR4 and sensitizes airway epithelial cells to endotoxin.

http://www.wikidata.org/entity/Q40625076

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Toll-like receptor 2 is upregulated by hog confinement dust in an IL-6-dependent manner in the airway epithelium Activation of the epidermal growth factor receptor by respiratory syncytial virus results in increased inflammation and delayed apoptosis Airway epithelial cell tolerance to Pseudomonas aeruginosa.Characterization of Toll-like receptors in primary lung epithelial cells: strong impact of the TLR3 ligand poly(I:C) on the regulation of Toll-like receptors, adaptor proteins and inflammatory response Respiratory epithelial cells require Toll-like receptor 4 for induction of human beta-defensin 2 by lipopolysaccharide Up-regulation of Toll-like receptors 2, 3 and 4 in allergic rhinitis.Intrinsic Cellular Defenses against Virus Infection by Antiviral Type I Interferon Respiratory epithelial cells orchestrate pulmonary innate immunity.TLR3 activation evokes IL-6 secretion, autocrine regulation of Stat3 signaling and TLR2 expression in human bronchial epithelial cells Diesel exhaust particles increase IL-1beta-induced human beta-defensin expression via NF-kappaB-mediated pathway in human lung epithelial cells An NF-κB-independent and Erk1/2-dependent mechanism controls CXCL8/IL-8 responses of airway epithelial cells to cadmium Innate immunity proteins and a new truncated form of SPLUNC1 in nasopharyngeal aspirates from infants with respiratory syncytial virus infection.Respiratory syncytial virus synergizes with Th2 cytokines to induce optimal levels of TARC/CCL17.Respiratory syncytial virus infection induces higher Toll-like receptor-3 expression and TNF-α production than human metapneumovirus infection Inhibition of Toll-like receptor 2-mediated interleukin-8 production in Cystic Fibrosis airway epithelial cells via the alpha7-nicotinic acetylcholine receptor Inhibiting toll-like receptor 4 signaling ameliorates pulmonary fibrosis during acute lung injury induced by lipopolysaccharide: an experimental study Respiratory viral infections in infants: causes, clinical symptoms, virology, and immunology.Pulmonary surfactant phosphatidylglycerol inhibits respiratory syncytial virus-induced inflammation and infection Differential role for TLR3 in respiratory syncytial virus-induced chemokine expression.Porcine reproductive and respiratory syndrome virus induces IL-1β production depending on TLR4/MyD88 pathway and NLRP3 inflammasome in primary porcine alveolar macrophages.Activation of interferon response through toll-like receptor 3 impacts viral pathogenesis and pulmonary toll-like receptor expression during respiratory syncytial virus and influenza infections in the cotton rat Sigmodon hispidus model.Immune responses and disease enhancement during respiratory syncytial virus infection Nucleoside diphosphate kinase and flagellin from Pseudomonas aeruginosa induce interleukin 1 expression via the Akt/NF-κB signaling pathways.Innate immune recognition of respiratory syncytial virus infection.Sensitization of human aortic endothelial cells to lipopolysaccharide via regulation of Toll-like receptor 4 by bacterial fimbria-dependent invasion.Chandipura virus infection in mice: the role of toll like receptor 4 in pathogenesis.The infectious march: the complex interaction between microbes and the immune system in asthma.Therapeutic targeting of respiratory syncytial virus G-protein.Toward primary prevention of asthma. Reviewing the evidence for early-life respiratory viral infections as modifiable risk factors to prevent childhood asthma Toll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus Infection Into the eye of the cytokine storm.Genetic variation in bactericidal/permeability-increasing protein influences the risk of developing rapid airflow decline after hematopoietic cell transplantation.House Dust Mite Allergens and the Induction of Monocyte Interleukin 1β Production That Triggers an IκBζ-Dependent Granulocyte Macrophage Colony-Stimulating Factor Release from Human Lung Epithelial Cells Respiratory syncytial virus infection provokes airway remodelling in allergen-exposed mice in absence of prior allergen sensitization.Mammalian Toll-like receptors: to immunity and beyond.Blockade of Interplay between IL-17A and Endoplasmic Reticulum Stress Attenuates LPS-Induced Lung Injury Pathogenesis of respiratory syncytial virus infection in the murine model Toll-like receptor 2 represses nonpilus adhesin-induced signaling in acute infections with the Pseudomonas aeruginosa pilA mutant.Effects of lipopolysaccharide and Mannheimia haemolytica leukotoxin on bovine lung microvascular endothelial cells and alveolar epithelial cells.Viral and host factors in human respiratory syncytial virus pathogenesis.

P2860

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P2860

Respiratory syncytial virus up-regulates TLR4 and sensitizes airway epithelial cells to endotoxin.

http://www.wikidata.org/entity/Q40625076

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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2003年论文

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2003年论文

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name

Respiratory syncytial virus up ...... epithelial cells to endotoxin.

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type

label

Respiratory syncytial virus up ...... epithelial cells to endotoxin.

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prefLabel

Respiratory syncytial virus up ...... epithelial cells to endotoxin.

@en

P1433

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P356

P1433

Journal of Biological Chemistry

P1476

Respiratory syncytial virus up ...... epithelial cells to endotoxin

@en

P2093

A Brent Carter

http://www.w3.org/2001/XMLSchema#string

Gary W Hunninghake

http://www.w3.org/2001/XMLSchema#string

Linda S Powers

http://www.w3.org/2001/XMLSchema#string

Martha M Monick

http://www.w3.org/2001/XMLSchema#string

Timur O Yarovinsky

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P2860

Interactions between genes and environmental factors in asthma and atopy: new developments

Innate immune recognition

Lipopolysaccharide rapidly traffics to and from the Golgi apparatus with the toll-like receptor 4-MD-2-CD14 complex in a process that is distinct from the initiation of signal transduction

Decreased expression of Toll-like receptor-4 and MD-2 correlates with intestinal epithelial cell protection against dysregulated proinflammatory gene expression in response to bacterial lipopolysaccharide

TLR4 and MD-2 expression is regulated by immune-mediated signals in human intestinal epithelial cells

Early childhood infectious diseases and the development of asthma up to school age: a birth cohort study

Environmental exposure to endotoxin and its relation to asthma in school-age children

Toll-like receptors and innate immunity

Human intestinal epithelial cells are broadly unresponsive to Toll-like receptor 2-dependent bacterial ligands: implications for host-microbial interactions in the gut.

Endotoxin, toll-like receptor 4, and the afferent limb of innate immunity.

P304

53035-53044

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scholarly article

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10.1074/JBC.M308093200

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52

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278

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Gunnar Gudmundsson

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2003-10-17T00:00:00Z

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14565959

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P921

Toll-like receptor