Jun N-terminal kinase activity and early growth-response factor-1 gene expression are down-regulated in Fanconi anemia group A lymphoblasts.
about
Elevated levels of STAT1 in Fanconi anemia group A lymphoblasts correlate with the cells' sensitivity to DNA interstrand crosslinking drugs.Impaired synthesis of heme oxygenase-1 in Fanconi anemia cells can be rescued by transfection of Fanconi wild-type cDNA.Gonadotropin-releasing hormone regulates expression of the DNA damage repair gene, Fanconi anemia A, in pituitary gonadotroph cellsFibroblasts from long-lived mutant mice show diminished ERK1/2 phosphorylation but exaggerated induction of immediate early genes.Elevated levels of IL-1beta in Fanconi anaemia group A patients due to a constitutively active phosphoinositide 3-kinase-Akt pathway are capable of promoting tumour cell proliferation.
P2860
Jun N-terminal kinase activity and early growth-response factor-1 gene expression are down-regulated in Fanconi anemia group A lymphoblasts.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Jun N-terminal kinase activity ...... i anemia group A lymphoblasts.
@en
type
label
Jun N-terminal kinase activity ...... i anemia group A lymphoblasts.
@en
prefLabel
Jun N-terminal kinase activity ...... i anemia group A lymphoblasts.
@en
P1433
P1476
Jun N-terminal kinase activity ...... ni anemia group A lymphoblasts
@en
P2093
Jose Antonio Casado
Jose Luis Fernandez-Luna
P304
P356
10.1182/BLOOD-2003-06-2091
P407
P577
2003-09-04T00:00:00Z