A coordinated cytotoxic effect of IFN-gamma and cross-reactive antibodies in the pathogenesis of Helicobacter pylori gastritis.
about
Regulation of gastric B cell recruitment is dependent on IL-17 receptor A signaling in a model of chronic bacterial infection.Systems modeling of the role of interleukin-21 in the maintenance of effector CD4+ T cell responses during chronic Helicobacter pylori infection.IL-17a and IL-22 Induce Expression of Antimicrobials in Gastrointestinal Epithelial Cells and May Contribute to Epithelial Cell Defense against Helicobacter pyloriHelicobacter pylori Resists the Antimicrobial Activity of Calprotectin via Lipid A Modification and Associated Biofilm Formation.
P2860
A coordinated cytotoxic effect of IFN-gamma and cross-reactive antibodies in the pathogenesis of Helicobacter pylori gastritis.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
A coordinated cytotoxic effect ...... Helicobacter pylori gastritis.
@en
type
label
A coordinated cytotoxic effect ...... Helicobacter pylori gastritis.
@en
prefLabel
A coordinated cytotoxic effect ...... Helicobacter pylori gastritis.
@en
P2093
P2860
P1433
P1476
A coordinated cytotoxic effect ...... Helicobacter pylori gastritis.
@en
P2093
Masaru Yoshida
Toshiyuki Itoh
Tsutomu Chiba
Yoshio Wakatsuki
P2860
P304
P356
10.1046/J.1523-5378.2003.00154.X
P577
2003-08-01T00:00:00Z