Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

about

HUS and atypical HUS.Complementopathies.In vitro evidence of complement activation in patients with sickle cell disease.

P2860

Q33441069-6559F37D-5A3A-42B2-AB48-7F1DA74949BB Q39142842-A416EFDA-AFBF-4D04-B3A7-79223C96C8BB Q47163783-A055561C-04F6-4B09-BF33-F5E0F28084C1

P2860

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

Item

http://www.wikidata.org/entity/Q40652002

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

@en

type

Item

label

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

@en

prefLabel

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

@en

P1476

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak

@en

P2093

Cozumel Pruette

http://www.w3.org/2001/XMLSchema#string

Darcy Weidemann

http://www.w3.org/2001/XMLSchema#string

Eleni Gavriilaki

http://www.w3.org/2001/XMLSchema#string

John J Strouse

http://www.w3.org/2001/XMLSchema#string

Lauren F Loeffler

http://www.w3.org/2001/XMLSchema#string

Robert A Brodsky

http://www.w3.org/2001/XMLSchema#string

Tammy M Brady

http://www.w3.org/2001/XMLSchema#string

P2860

Alternative pathway of complement in children with diarrhea-associated hemolytic uremic syndrome.

Eculizumab in severe Shiga-toxin-associated HUS

Alternative pathway activation of complement by Shiga toxin promotes exuberant C3a formation that triggers microvascular thrombosis

Shiga toxin activates complement and binds factor H: evidence for an active role of complement in hemolytic uremic syndrome

A murine model of HUS: Shiga toxin with lipopolysaccharide mimics the renal damage and physiologic response of human disease.

Decrease of thrombomodulin contributes to the procoagulant state of endothelium in hemolytic uremic syndrome.

Spontaneous hemolytic uremic syndrome triggered by complement factor H lacking surface recognition domains.

Shiga toxin-2 results in renal tubular injury but not thrombotic microangiopathy in heterozygous factor H-deficient mice.

Validation of treatment strategies for enterohaemorrhagic Escherichia coli O104:H4 induced haemolytic uraemic syndrome: case-control study

Lack of the lectin-like domain of thrombomodulin worsens Shiga toxin-associated hemolytic uremic syndrome in mice.

P31

scholarly article

P356

10.21767/2472-5056.100011

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-05-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27413789

http://www.w3.org/2001/XMLSchema#string

P932

4940046

http://www.w3.org/2001/XMLSchema#string

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

about

P2860

P2860

Typical Hus: Evidence of Acute Phase Complement Activation from a Daycare Outbreak.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P31

P356

P433

P478

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P31

P356

P433

P478

P577

P698

P932