Activation of endothelial nitric-oxide synthase by tumor necrosis factor-alpha: a novel pathway involving sequential activation of neutral sphingomyelinase, phosphatidylinositol-3' kinase, and Akt.
about
Nitrosation-dependent caveolin 1 phosphorylation, ubiquitination, and degradation and its association with idiopathic pulmonary arterial hypertensionArgininosuccinate synthase: at the center of arginine metabolismNitric oxide-sphingolipid interplays in plant signalling: a new enigma from the Sphinx?Roles and regulation of neutral sphingomyelinase-2 in cellular and pathological processesResistance to endotoxic shock in endothelial nitric-oxide synthase (eNOS) knock-out mice: a pro-inflammatory role for eNOS-derived no in vivoRyanodine receptor leak mediated by caspase-8 activation leads to left ventricular injury after myocardial ischemia-reperfusionTumor necrosis factor α decreases nitric oxide synthase type 3 expression primarily via Rho/Rho kinase in the thick ascending limbRegulation of obesity and insulin resistance by nitric oxideGPR40/FFA1 and neutral sphingomyelinase are involved in palmitate-boosted inflammatory response of microvascular endothelial cells to LPS.Impact of exposure to low concentrations of nitric oxide on protein profile in murine and human pancreatic islet cellsRecent advances in the immunobiology of ceramide.Adipokines and the blood-brain barrierFenretinide inhibited de novo ceramide synthesis and proinflammatory cytokines induced by Aggregatibacter actinomycetemcomitans.The role of nitric oxide in mediating tumour blood flow.Nitric oxide and sphingolipids: mechanisms of interaction and role in cellular pathophysiology.Increased intestinal macromolecular permeability and urine nitrite excretion associated with liver cirrhosis with ascitesBiological roles of Acid and neutral sphingomyelinases and their regulation by nitric oxide.Regulation of NADPH oxidases in skeletal muscle.Sphingomyelinase causes endothelium-dependent vasorelaxation through endothelial nitric oxide production without cytosolic Ca(2+) elevation.In human endothelial cells rapamycin causes mTORC2 inhibition and impairs cell viability and function.TNF-alpha potentiates protein-tyrosine nitration through activation of NADPH oxidase and eNOS localized in membrane rafts and caveolae of bovine aortic endothelial cells.Estrogen receptor activation protects against TNF-α-induced endothelial dysfunction.Apigenin inhibits the TNFα-induced expression of eNOS and MMP-9 via modulating Akt signalling through oestrogen receptor engagement.Nitric Oxide Generated by Tumor-Associated Macrophages Is Responsible for Cancer Resistance to Cisplatin and Correlated With Syntaxin 4 and Acid Sphingomyelinase Inhibition.
P2860
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P2860
Activation of endothelial nitric-oxide synthase by tumor necrosis factor-alpha: a novel pathway involving sequential activation of neutral sphingomyelinase, phosphatidylinositol-3' kinase, and Akt.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Activation of endothelial nitr ...... ylinositol-3' kinase, and Akt.
@en
type
label
Activation of endothelial nitr ...... ylinositol-3' kinase, and Akt.
@en
prefLabel
Activation of endothelial nitr ...... ylinositol-3' kinase, and Akt.
@en
P2093
P50
P356
P1476
Activation of endothelial nitr ...... dylinositol-3' kinase, and Akt
@en
P2093
Rico Barsacchi
Salvador Moncada
Stefania Bulotta
P304
P356
10.1124/MOL.63.4.886
P577
2003-04-01T00:00:00Z