Diesel exhaust particles upregulate eotaxin gene expression in human bronchial epithelial cells via nuclear factor-kappa B-dependent pathway.
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Combustion of dried animal dung as biofuel results in the generation of highly redox active fine particulatesImpact of air pollution on allergic diseasesEmerging mechanistic targets in lung injury induced by combustion-generated particlesRegulatory T cells protect fine particulate matter-induced inflammatory responses in human umbilical vein endothelial cellsActivation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering MechanismsNrf2 is a protective factor against oxidative stresses induced by diesel exhaust particle in allergic asthmaMouse models to unravel the role of inhaled pollutants on allergic sensitization and airway inflammationResponses of airway epithelium to environmental injury: role in the induction phase of childhood asthmaRole of neprilysin in airway inflammation induced by diesel exhaust emissions.The generation of diesel exhaust particle aerosols from a bulk source in an aerodynamic size range similar to atmospheric particles.Exacerbation of allergic inflammation in mice exposed to diesel exhaust particles prior to viral infection.Airway inflammatory response to diesel exhaust generated at urban cycle running conditions.Nrf2 Regulates the Risk of a Diesel Exhaust Inhalation-Induced Immune Response during Bleomycin Lung Injury and Fibrosis in Mice.Chronic traffic pollution exposure is associated with eosinophilic, but not neutrophilic inflammation in older adult asthmatics.Ambient vapor samples activate the Nrf2-ARE pathway in human bronchial epithelial BEAS-2B cellsAnti-inflammatory effects of limonene from yuzu (Citrus junos Tanaka) essential oil on eosinophils.The effects of oxidative stress induced by prolonged low-dose diesel exhaust particle exposure on the generation of allergic airway inflammation differ between BALB/c and C57BL/6 mice.Airway inflammatory responses to oxidative stress induced by prolonged low-dose diesel exhaust particle exposure from birth differ between mouse BALB/c and C57BL/6 strains.Responses of differentiated primary human lung epithelial cells to exposure to diesel exhaust at an air-liquid interface.In vitro effects of nanoparticle-rich diesel exhaust particles on splenic mononuclear cells.
P2860
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P2860
Diesel exhaust particles upregulate eotaxin gene expression in human bronchial epithelial cells via nuclear factor-kappa B-dependent pathway.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Diesel exhaust particles upreg ...... tor-kappa B-dependent pathway.
@en
type
label
Diesel exhaust particles upreg ...... tor-kappa B-dependent pathway.
@en
prefLabel
Diesel exhaust particles upreg ...... tor-kappa B-dependent pathway.
@en
P2093
P2860
P1476
Diesel exhaust particles upreg ...... tor-kappa B-dependent pathway.
@en
P2093
Hajime Takizawa
Hitoshi Okazaki
Isamu Sugawara
Kazuhiko Nakahara
Kazuhiko Yamamoto
Kouji Matsushima
Masaru Sagai
Masashi Desaki
Mitsuru Tanaka
Shin Kawasaki
P2860
P304
P356
10.1152/AJPLUNG.00358.2002
P577
2003-02-07T00:00:00Z