A futile metabolic cycle activated in adipocytes by antidiabetic agents. - Wikidata - awgldk - TriplyDB

A futile metabolic cycle activated in adipocytes by antidiabetic agents.

A futile metabolic cycle activated in adipocytes by antidiabetic agents.

http://www.wikidata.org/entity/Q40700524

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Cytosolic aspartate aminotransferase, a new partner in adipocyte glyceroneogenesis and an atypical target of thiazolidinedione Moonlighting function of glycerol kinase causes systems-level changes in rat hepatoma cells Implication of the Pro12Ala polymorphism of the PPAR-gamma 2 gene in type 2 diabetes and obesity in the French population.Modulation of the transcriptional activity of peroxisome proliferator-activated receptor gamma by protein-protein interactions and post-translational modifications Structure and non-essential function of glycerol kinase inPlasmodium falciparumblood stages Obesity is associated with macrophage accumulation in adipose tissue PPARγ as a therapeutic target to rescue mitochondrial function in neurological disease The cell biology of fat expansion C13orf31 (FAMIN) is a central regulator of immunometabolic function Identification of a novel mitochondrial protein ("mitoNEET") cross-linked specifically by a thiazolidinedione photoprobe 'Striking the Right Balance' in Targeting PPARgamma in the Metabolic Syndrome: Novel Insights from Human Genetic Studies Nutrigenomics of body weight regulation: a rationale for careful dissection of individual contributors Transcriptomic and network component analysis of glycerol kinase in skeletal muscle using a mouse model of glycerol kinase deficiency PPARalpha governs glycerol metabolism Corepressors selectively control the transcriptional activity of PPARgamma in adipocytes Metabolic control of muscle mitochondrial function and fatty acid oxidation through SIRT1/PGC-1alpha Expression of glycerokinase in brown adipose tissue is stimulated by the sympathetic nervous system PPARgamma regulates adipose triglyceride lipase in adipocytes in vitro and in vivo.Long-term ritonavir exposure increases fatty acid and glycerol recycling in 3T3-L1 adipocytes as compensatory mechanisms for increased triacylglycerol hydrolysis.Aquaporin 7 deficiency is associated with development of obesity through activation of adipose glycerol kinase.Obesity and metabolic perturbations after loss of aquaporin 7, the adipose glycerol transporter.Memory CD8(+) T cells use cell-intrinsic lipolysis to support the metabolic programming necessary for development.Glycerol kinase deficiency alters expression of genes involved in lipid metabolism, carbohydrate metabolism, and insulin signaling.Global metabolic effects of glycerol kinase overexpression in rat hepatoma cells.Involvement of inducible 6-phosphofructo-2-kinase in the anti-diabetic effect of peroxisome proliferator-activated receptor gamma activation in mice.Apelin-13 decreases lipid storage in hypertrophic adipocytes in vitro through the upregulation of AQP7 expression by the PI3K signaling pathway Trans-10,cis-12 CLA increases adipocyte lipolysis and alters lipid droplet-associated proteins: role of mTOR and ERK signaling.Transcriptional targets in adipocyte biology Bone marrow fat has brown adipose tissue characteristics, which are attenuated with aging and diabetes.Combined transcriptomic-(1)H NMR metabonomic study reveals that monoethylhexyl phthalate stimulates adipogenesis and glyceroneogenesis in human adipocytes.DEPTOR cell-autonomously promotes adipogenesis, and its expression is associated with obesity.RIP140 represses the "brown-in-white" adipocyte program including a futile cycle of triacylglycerol breakdown and synthesis.PPAR gamma and human metabolic disease Phosphoenolpyruvate carboxykinase (Pck1) helps regulate the triglyceride/fatty acid cycle and development of insulin resistance in mice.Revisiting PPARγ as a target for the treatment of metabolic disorders Function of phosphoenolpyruvate carboxykinase in mammary gland epithelial cells.Aquaglyceroporins serve as metabolic gateways in adiposity and insulin resistance control.PPARgamma and metabolism: insights from the study of human genetic variants.Hypoxia induces a lipogenic cancer cell phenotype via HIF1α-dependent and -independent pathways.Mathematical modeling of the insulin signal transduction pathway for prediction of insulin sensitivity from expression data.

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P2860

A futile metabolic cycle activated in adipocytes by antidiabetic agents.

http://www.wikidata.org/entity/Q40700524

description

2002 nî lūn-bûn

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2002年の論文

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2002年論文

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2002年论文

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name

A futile metabolic cycle activated in adipocytes by antidiabetic agents.

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label

A futile metabolic cycle activated in adipocytes by antidiabetic agents.

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A futile metabolic cycle activated in adipocytes by antidiabetic agents.

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A futile metabolic cycle activated in adipocytes by antidiabetic agents.

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Christopher B Newgard

http://www.w3.org/2001/XMLSchema#string

Claire M Steppan

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Hong-Ping Guan

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Mette Valentin Jensen

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Mitchell A Lazar

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Yong Li

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P2860

Oxidized LDL regulates macrophage gene expression through ligand activation of PPARgamma

Fatty acids and eicosanoids regulate gene expression through direct interactions with peroxisome proliferator-activated receptors alpha and gamma

PPAR gamma is required for the differentiation of adipose tissue in vivo and in vitro

X-linked glycerol kinase deficiency in the mouse leads to growth retardation, altered fat metabolism, autonomous glucocorticoid secretion and neonatal death

PPAR gamma 2 regulates adipose expression of the phosphoenolpyruvate carboxykinase gene

15-Deoxy-delta 12, 14-prostaglandin J2 is a ligand for the adipocyte determination factor PPAR gamma

The hormone resistin links obesity to diabetes

Obesity as a medical problem

mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer

A prostaglandin J2 metabolite binds peroxisome proliferator-activated receptor gamma and promotes adipocyte differentiation

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