about
Vanilloid receptor 1 expression in the rat urinary tract.Can the adrenergic system be implicated in the pathophysiology of bladder pain syndrome/interstitial cystitis? A clinical and experimental studyThe role of brain-derived neurotrophic factor (BDNF) in the development of neurogenic detrusor overactivity (NDO).Neurotrophins in the lower urinary tract: becoming of ageThe ERK 1 and 2 pathway in the nervous system: from basic aspects to possible clinical applications in pain and visceral dysfunction.Nerve growth factor in bladder dysfunction: contributing factor, biomarker, and therapeutic target.Neurotrophins as regulators of urinary bladder function.Neurotrophins in bladder function: what do we know and where do we go from here?Biomarkers in lower urinary tract symptoms/overactive bladder: a critical overview.Biomarkers of spinal cord injury and ensuing bladder dysfunction.Urinary neurotrophic factors in healthy individuals and patients with overactive bladder.Rat detrusor overactivity induced by chronic spinalization can be abolished by a transient receptor potential vanilloid 1 (TRPV1) antagonist.An update on neurotrophins.Nerve growth factor regulates galanin and c-jun overexpression occurring in dorsal root ganglion cells after intravesical resiniferatoxin application.Co-administration of transient receptor potential vanilloid 4 (TRPV4) and TRPV1 antagonists potentiate the effect of each drug in a rat model of cystitis.Impairment of sensory afferents by intrathecal administration of botulinum toxin A improves neurogenic detrusor overactivity in chronic spinal cord injured rats.Distribution of the high-affinity binding site and intracellular target of botulinum toxin type A in the human bladder.ACTH modulates ERK phosphorylation in the adrenal gland in a time-dependent manner.Increased spinal cord phosphorylation of extracellular signal-regulated kinases mediates micturition overactivity in rats with chronic bladder inflammation.GRC-6211, a new oral specific TRPV1 antagonist, decreases bladder overactivity and noxious bladder input in cystitis animal models.Intrathecal delivery of resiniferatoxin (RTX) reduces detrusor overactivity and spinal expression of TRPV1 in spinal cord injured animals.Effect of onabotulinumtoxinA on intramural parasympathetic ganglia: an experimental study in the guinea pig bladder.Trigonal injection of botulinum toxin A in patients with refractory bladder pain syndrome/interstitial cystitis.Urinary Neurotrophin Levels Increase in Women With Stress Urinary Incontinence After a Midurethral Sling Procedure.Transient receptor potential vanilloid subfamily 1 is essential for the generation of noxious bladder input and bladder overactivity in cystitis.Ulcerative and nonulcerative forms of bladder pain syndrome/interstitial cystitis do not differ in symptom intensity or response to onabotulinum toxin A.Spinal cord injury and bladder dysfunction: new ideas about an old problem.Spread of onabotulinumtoxinA after bladder injection. Experimental study using the distribution of cleaved SNAP-25 as the marker of the toxin actionReply from Authors re: Prokar Dasgupta. Volume Matters: Bladder Injections of Botulinum Toxin Type A. Eur Urol 2012;61:1185–6817 URINARY NEUROTROPHIC FACTORS IN BLADDER PAIN SYNDROME/INTERSTITIAL CYSTITISTransient receptor potential vanilloid 1 mediates nerve growth factor-induced bladder hyperactivity and noxious inputAnimal Models of Cystitis813 INCREASED SYMPATHETIC ACTIVITY ENHANCES BLADDER HYPERACTIVITY AND TRIGGERS BLADDER PAIN1677 INTRA-TRIGONAL INJECTION OF BOTULINUM TOXIN A IN PATIENTS WITH REFRACTORY BLADDER PAIN SYNDROME DECREASES URINARY NEUROTROPHINS AND IMPROVES LOWER URINARY TRACT SYMPTOMS191 EFFECT OF BOTULINUM TOXIN TYPE A ON INTRAMURAL PARASYMPATHETIC GANGLIA OF THE GUINEA-PIG BLADDERHIGH AFFINITY BINDING SITES FOR BOTULINUM TOXIN TYPE A IN THE URINARY BLADDER: DISTRIBUTION AND NEUROCHEMISTRYTHE ORAL TRPV1 ANTAGONIST GRC 6211 REDUCES BLADDER OVERACTIVITY AND NOXIOUS BLADDER INPUT IN CYSTITISEffects of early intravesical administration of resiniferatoxin to spinal cord-injured rats in neurogenic detrusor overactivity
P50
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