Toll-like receptor 2 is required for optimal control of Listeria monocytogenes infection
about
Function of Nod-like receptors in microbial recognition and host defenseCytolysin-dependent escape of the bacterium from the phagosome is required but not sufficient for induction of the Th1 immune response against Listeria monocytogenes infection: distinct role of Listeriolysin O determined by cytolysin gene replacementThe tyrosine kinase Btk regulates the macrophage response to Listeria monocytogenes infectionTLR2 and RIP2 pathways mediate autophagy of Listeria monocytogenes via extracellular signal-regulated kinase (ERK) activationMyD88-mediated signals induce the bactericidal lectin RegIII gamma and protect mice against intestinal Listeria monocytogenes infectionInterplay between CD8α+ dendritic cells and monocytes in response to Listeria monocytogenes infection attenuates T cell responsesA critical role for peptidoglycan N-deacetylation in Listeria evasion from the host innate immune system.Toll-like receptor 2- and MyD88-dependent phosphatidylinositol 3-kinase and Rac1 activation facilitates the phagocytosis of Listeria monocytogenes by murine macrophages.The apoptotic response to pneumolysin is Toll-like receptor 4 dependent and protects against pneumococcal diseaseBoth TLR2 and TRIF contribute to interferon-β production during Listeria infectionWhen Toll-like receptor and T-cell receptor signals collide: a mechanism for enhanced CD8 T-cell effector function.Formylpeptide receptors are critical for rapid neutrophil mobilization in host defense against Listeria monocytogenes.Membrane damage during Listeria monocytogenes infection triggers a caspase-7 dependent cytoprotective responseBacterial programming of host responses: coordination between type I interferon and cell death.Neonatal innate immunity to infectious agents.Growth temperature-dependent contributions of response regulators, σB, PrfA, and motility factors to Listeria monocytogenes invasion of Caco-2 cells.Innate immune recognition and inflammasome activation in listeria monocytogenes infection.Nitric oxide and redox mechanisms in the immune response.The early phase transcriptome of bovine monocyte-derived macrophages infected with Staphylococcus aureus in vitro.Priming of protective anti-Listeria monocytogenes memory CD8+ T cells requires a functional SecA2 secretion systemMembrane tumor necrosis factor confers partial protection to Listeria infection.Peptidoglycan induces necrosis and regulates cytokine production in murine trophoblast stem cells.Indoleamine 2,3-dioxygenase-expressing dendritic cells form suppurative granulomas following Listeria monocytogenes infectionInnate immune response to Streptococcus pyogenes depends on the combined activation of TLR13 and TLR2.Mutations of the Listeria monocytogenes peptidoglycan N-deacetylase and O-acetylase result in enhanced lysozyme sensitivity, bacteriolysis, and hyperinduction of innate immune pathways.Cytosolic entry controls CD8+-T-cell potency during bacterial infection.Regulation of L-type Voltage Gated Calcium Channel CACNA1S in Macrophages upon Mycobacterium tuberculosis Infection.Deception point: peptidoglycan modification as a means of immune evasion.Listeria monocytogenes surface proteins: from genome predictions to function.An NLRP7-containing inflammasome mediates recognition of microbial lipopeptides in human macrophagesInvolvement of the AIM2, NLRC4, and NLRP3 inflammasomes in caspase-1 activation by Listeria monocytogenes.Growth of Yersinia pseudotuberculosis in mice occurs independently of Toll-like receptor 2 expression and induction of interleukin-10.Novel bacterial lipoprotein structures conserved in low-GC content gram-positive bacteria are recognized by Toll-like receptor 2.The Role of TLR2 in Infection and ImmunityListeria monocytogenes strain-specific impairment of the TetR regulator underlies the drastic increase in cyclic di-AMP secretion and beta interferon-inducing ability.CCR-2 neutralization augments murine fresh BMC activation by Staphylococcus aureus via two distinct mechanisms: at the level of ROS production and cytokine response.Lead and immune function.Activation of cytosolic phospholipase A2alpha in resident peritoneal macrophages by Listeria monocytogenes involves listeriolysin O and TLR2.Variation in the TLR10/TLR1/TLR6 locus is the major genetic determinant of interindividual difference in TLR1/2-mediated responsesToll-like receptor (TLR) 2 mediates inflammatory responses to oligomerized RrgA pneumococcal pilus type 1 protein.
P2860
Q24647448-4AAB1452-8420-4564-B822-46485BB6004BQ24675620-B36C34CA-5845-4972-AB45-AED691B1FB5AQ28504622-40BF1509-DC66-42E2-9845-E97916EA6E2BQ28507939-837FCA78-F636-428F-8D84-AED5E4B4A343Q28585035-225362FE-F5F0-424E-AB2C-019E04029C9DQ28740555-FD2A5498-4745-4340-8ABA-2E8005A78AD6Q33268810-D35FA773-83DD-4AC9-A1CF-3E56F32AEA61Q33877279-C0160944-8715-4810-A93C-3331EA12A1A0Q34033179-2AF973E2-65A3-49F0-8B15-F1F6812D8A83Q34202860-8028767F-C061-4E1C-ADAD-1F5505C434E9Q34310528-90775202-E8F3-4ADF-9B6F-3929C9CA459FQ34310804-B3FF4823-D216-40FE-9143-12A31BAF5963Q34341346-34580ADE-D2D5-4719-9EC1-A0477CD9786CQ34414006-A21013F2-5E5C-422B-9139-15AFE8DA82F6Q34492674-FDCDA232-804A-4325-B414-0C75C1714B47Q34814672-5FE7AEBD-C3F8-4CDA-A73F-81C6745E9F5BQ34983641-FA890AF8-0B87-4458-84EC-BBB639A15894Q35001939-9F59C28E-6DD9-4BEA-8882-AD487397C90EQ35068422-E1E7B36F-E34C-45D4-B3B7-3AB0B452D07AQ35075744-3B080F66-F3BB-4AEE-8EAA-50D60FCC6B82Q35094506-24145B55-06BD-444C-BC4F-2DA65B3FE787Q35109234-4CD1FFDE-6F12-42B2-840A-7E93D9E9B842Q35131219-35D50387-A476-40EE-A837-5155066A56E1Q35164770-48C6356C-DF21-4EEC-98E4-D99ECDE24A55Q35191858-86E90CEC-7D1A-43E1-8D4A-18F17D1EB57CQ35217477-BE78F502-A88D-4843-AF4C-9617DAD3C21CQ35542683-D580AA54-85CF-4276-8652-780ABB2BB3B0Q35612173-522F2FF7-98C0-499F-81D8-CEFC61AD4B1AQ35856683-3DA9F359-2E3F-4A27-8EBB-2E620C5B5D5CQ35860827-C66E9442-2233-4FB3-8CC4-248E9D31A12EQ35876024-4E6D9E5F-30D8-48D4-8F48-6D860DDC4F65Q35913579-62D8DADD-2DAF-4DAA-B525-0C721910D537Q35921603-2F49E6F6-F032-4B39-8F59-1186C88372AEQ35927566-C318BE0D-BD27-4737-85E5-27FD2BCFF5EEQ36156009-C4A07DD2-9F4D-4D68-B283-56F9EBFB1318Q36346773-0865FC05-7AE5-4B32-9379-BDCBB02A3E58Q36523480-E8D9A124-5358-4A56-A45D-C183BEB2ECFFQ36543744-1200FA40-384A-4115-8D0B-CA74C313BC0EQ36561773-AFD2D8A8-A0D6-4F03-8BD9-BAC1A26ECFD2Q36562085-FC849C1C-F05E-4724-8285-29F7A633C418
P2860
Toll-like receptor 2 is required for optimal control of Listeria monocytogenes infection
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Toll-like receptor 2 is required for optimal control of Listeria monocytogenes infection
@en
type
label
Toll-like receptor 2 is required for optimal control of Listeria monocytogenes infection
@en
prefLabel
Toll-like receptor 2 is required for optimal control of Listeria monocytogenes infection
@en
P2093
P2860
P1476
Toll-like receptor 2 is required for optimal control of Listeria monocytogenes infection
@en
P2093
David Torres
Franck Bihl
François Erard
Isabelle Maillet
Mathieu Barrier
Valerie J F Quesniaux
P2860
P304
P356
10.1128/IAI.72.4.2131-2139.2004
P407
P577
2004-04-01T00:00:00Z