HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis. - Wikidata - awgldk - TriplyDB

HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

http://www.wikidata.org/entity/Q40720204

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Hepatitis C virus non-structural protein NS5A interacts with FKBP38 and inhibits apoptosis in Huh7 hepatoma cells Phosphorylation of hepatitis C virus nonstructural protein 5A modulates its protein interactions and viral RNA replication Hepatitis C Virus Infection Induces Autophagy as a Prosurvival Mechanism to Alleviate Hepatic ER-Stress Response Disruption of Bcl-2 and Bcl-xL by viral proteins as a possible cause of cancer Classical swine fever virus NS5A protein changed inflammatory cytokine secretion in porcine alveolar macrophages by inhibiting the NF-κB signaling pathway Hepatitis C virus NS5A-mediated activation of phosphoinositide 3-kinase results in stabilization of cellular beta-catenin and stimulation of beta-catenin-responsive transcription Identification of Residues Required for RNA Replication in Domains II and III of the Hepatitis C Virus NS5A Protein Hepatitis C Virus Infection Induces Apoptosis through a Bax-Triggered, Mitochondrion-Mediated, Caspase 3-Dependent Pathway Activation of Ribosomal RNA Transcription by Hepatitis C Virus Involves Upstream Binding Factor Phosphorylation via Induction of Cyclin D1 Hepatitis C virus and hepatocarcinogenesis Quantitative Proteomics Analysis of the Hepatitis C Virus Replicon High-Permissive and Low-Permissive Cell Lines Hepatitis C Virus Indirectly Disrupts DNA Damage-Induced p53 Responses by Activating Protein Kinase R.Induction of p53, p21 and apoptosis by silencing the NF90/NF45 complex in human papilloma virus-transformed cervical carcinoma cells.Ribavirin resistance in hepatitis C virus replicon-containing cell lines conferred by changes in the cell line or mutations in the replicon RNA.The oncogenic role of NS5A of hepatitis C virus is mediated by up-regulation of survivin gene expression in the hepatocellular cell through p53 and NF-κB pathways.Hepatitis C virus NS5A activates the mammalian target of rapamycin (mTOR) pathway, contributing to cell survival by disrupting the interaction between FK506-binding protein 38 (FKBP38) and mTOR Structural and molecular basis of interaction of HCV non-structural protein 5A with human casein kinase 1α and PKR.Infection of human cancer cells with myxoma virus requires Akt activation via interaction with a viral ankyrin-repeat host range factor.Viral hepatocarcinogenesis.Hepatitis C virus NS2 protein inhibits DNA damage pathway by sequestering p53 to the cytoplasm.Viral carcinogenesis: factors inducing DNA damage and virus integration Effect of hepatitis C infection on HIV-induced apoptosis Downregulation of Gadd45beta expression by hepatitis C virus leads to defective cell cycle arrest.TP53 and MDM2 gene polymorphisms and risk of hepatocellular carcinoma among Italian patients.Oncogenic potential of hepatitis C virus proteins Should NS5A inhibitors serve as the scaffold for all-oral anti-HCV combination therapies?Virus-host cell interactions during hepatitis C virus RNA replication: impact of polyprotein expression on the cellular transcriptome and cell cycle association with viral RNA synthesis.Tumor suppressors, chromosomal instability, and hepatitis C virus-associated liver cancer.Hepatitis C virus infection: establishment of chronicity and liver disease progression FUSE Binding Protein 1 Facilitates Persistent Hepatitis C Virus Replication in Hepatoma Cells by Regulating Tumor Suppressor p53.Hepatocellular carcinoma: role of hepatitis B and hepatitis C viruses proteins in hepatocarcinogenesis.Modulation of apoptosis as a target for liver disease.Hepatitis C virus and cellular stress response: implications to molecular pathogenesis of liver diseases.HCV NS5A abrogates p53 protein function by interfering with p53-DNA binding.Hepatitis C virus-related hepatocellular carcinoma: An insight into molecular mechanisms and therapeutic strategies Ankyrin Repeat Domain 1 is Up-regulated During Hepatitis C Virus Infection and Regulates Hepatitis C Virus Entry.Virus-specific mechanisms of carcinogenesis in hepatitis C virus associated liver cancer Phosphorylation drives an apoptotic protein to activate antiapoptotic genes: paradigm of influenza A matrix 1 protein function.How Hepatitis C Virus Leads to Hepatocellular Carcinoma: A Network-Based Study.Hepatitis C virus infection and apoptosis

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P2860

HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

http://www.wikidata.org/entity/Q40720204

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HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

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HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

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HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

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HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis.

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Full-Young Chang

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Jaw-Ching Wu

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Keng-Hsin Lan

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Masao Omata

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Meei-Ling Sheu

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Naoya Kato

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Sang-Hue Yen

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Shinn-Jang Hwang

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Shiow-Yi Chen

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Shou-Dong Lee

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P2860

Human TAFII31 protein is a transcriptional coactivator of the p53 protein

Genetic organization and diversity of the hepatitis C virus

Replication of subgenomic hepatitis C virus RNAs in a hepatoma cell line

Gene mapping of the putative structural region of the hepatitis C virus genome by in vitro processing analysis

Efficient selection for high-expression transfectants with a novel eukaryotic vector

Molecular cloning of the human hepatitis C virus genome from Japanese patients with non-A, non-B hepatitis

An assay for circulating antibodies to a major etiologic virus of human non-A, non-B hepatitis

p53, the cellular gatekeeper for growth and division

Hepatitis C virus infection is associated with the development of hepatocellular carcinoma

Apoptosis in the pathogenesis and treatment of disease

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