Murine cutaneous leishmaniasis: disease patterns in intact and nude mice of various genotypes and examination of some differences between normal and infected macrophages.
about
Leishmaniasis in beige miceTH1 and TH2 T-cell subsets are differentially activated by macrophages and B cells in murine leishmaniasis.Interleukin-4 but not gamma interferon production correlates with the severity of murine cutaneous leishmaniasis.Gamma interferon response in secondary Leishmania major infection: role of CD8+ T cellsOverexpression of a single Leishmania major gene enhances parasite infectivity in vivo and in vitroCompensation for decreased expression of B7 molecules on Leishmania infantum-infected canine macrophages results in restoration of parasite-specific T-cell proliferation and gamma interferon production.Persistence of virulent Leishmania major in murine cutaneous leishmaniasis: a possible hazard for the hostCharacterization of Leishmania major antigen-liposomes that protect BALB/c mice against cutaneous leishmaniasis.Spontaneous recovery of pathogenicity by Leishmania major hsp100-/- alters the immune response in mice.Expansion of gamma delta+ T cells in BALB/c mice infected with Leishmania major is dependent upon Th2-type CD4+ T cells.Protective vaccination with promastigote surface antigen 2 from Leishmania major is mediated by a TH1 type of immune responseTreatment of experimental cutaneous leishmaniasis with liposome-intercalated amphotericin B.Tumor necrosis factor alpha (TNF-alpha) and TNF-beta and their receptors in experimental cutaneous leishmaniasis.Expansion of gamma interferon-producing CD8+ T cells following secondary infection of mice immune to Leishmania major.Reduced pathology following infection with transgenic Leishmania major expressing murine CD40 ligand.Visceral spreading depletion of thymus-dependent regions and amyloidosis in mice and hamsters infected intradermally with Leishmania isolated from Sudanese cutaneous leishmaniasisStage-specific, strain-specific, and cross-reactive antigens of Leishmania species identified by monoclonal antibodiesVisceral Leishmania tropica infection of BALB/c mice: cellular analysis of in vitro unresponsiveness to sheep erythrocytes.Immunological regulation of experimental cutaneous leishmaniasis. IV. Prophylactic effect of sublethal irradiation as a result of abrogation of suppressor T cell generation in mice genetically susceptible to Leishmania tropica.Advantages of measuring changes in the number of viable parasites in murine models of experimental cutaneous leishmaniasis.Cutaneous leishmaniasis. The defect in T cell influx in BALB/c mice.The dynamics of granuloma formation in experimental visceral leishmaniasis.T cell and non-T cell compartments can independently determine resistance to Leishmania major.Failure of hamster macrophages to discriminate between infective and noninfective promastigotes of Leishmania donovani during attachment in vitroLeishmania major Hsp100 is required chiefly in the mammalian stage of the parasiteCutaneous leishmaniasis in the dorsal skin of hamsters: a useful model for the screening of antileishmanial drugsStudy of Leishmania major-infected macrophages by use of lipophosphoglycan-specific monoclonal antibodiesEvidence of T-cell recognition in mice of a purified lipophosphoglycan from Leishmania majorEffect of T-lymphocyte suppression on the parasite burden in Leishmania major-infected, genetically susceptible BALB/c miceSuppression of pathogenesis in cutaneous leishmaniasis by UV irradiation.Pathophysiology of experimental leishmaniasis: pattern of development of metastatic disease in the susceptible hostSpecific suppressor T cells for delayed-type hypersensitivity in susceptible mice immunized against cutaneous leishmaniasisResistance to cutaneous leishmaniasis: acquired ability of the host to kill parasites at the site of infection.Immunization with Leishmania receptor for macrophages protects mice against cutaneous leishmaniasis.Fate of Chlamydia trachomatis in human monocytes and monocyte-derived macrophages.Development of topical treatment for cutaneous leishmaniasis caused by Leishmania major in experimental animals.Interactions between murine macrophages and obligate intracellular protozoa.The kinetoplastid membrane protein 11 of Leishmania donovani and African trypanosomes is a potent stimulator of T-lymphocyte proliferation.The scientific challenge of Langerhans cell histiocytosis.Leishmania (Viannia) panamensis-induced cutaneous leishmaniasis in Balb/c mice: pathology
P2860
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P2860
Murine cutaneous leishmaniasis: disease patterns in intact and nude mice of various genotypes and examination of some differences between normal and infected macrophages.
description
1979 nî lūn-bûn
@nan
1979年の論文
@ja
1979年論文
@yue
1979年論文
@zh-hant
1979年論文
@zh-hk
1979年論文
@zh-mo
1979年論文
@zh-tw
1979年论文
@wuu
1979年论文
@zh
1979年论文
@zh-cn
name
Murine cutaneous leishmaniasis ...... rmal and infected macrophages.
@en
type
label
Murine cutaneous leishmaniasis ...... rmal and infected macrophages.
@en
prefLabel
Murine cutaneous leishmaniasis ...... rmal and infected macrophages.
@en
P2093
P1476
Murine cutaneous leishmaniasis ...... rmal and infected macrophages.
@en
P2093
P577
1979-02-01T00:00:00Z