Smad3 -signalling and Th2 cytokines in normal mouse airways and in a mouse model of asthma.
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The genetic and epigenetic landscapes of the epithelium in asthmaA large-scale, consortium-based genomewide association study of asthmaSurfactant protein D attenuates sub-epithelial fibrosis in allergic airways disease through TGF-βA protective role for periostin and TGF-β in IgE-mediated allergy and airway hyperresponsivenessTGF-beta can leave you breathlessThe interaction between mother and fetus and the development of allergic asthma.Genetic effects of multiple asthma loci identified by genomewide association studies on asthma and spirometric indices.An inhalation model of airway allergic response to inhalation of environmental Aspergillus fumigatus conidia in sensitized BALB/c mice.CTLA4Ig inhibits effector T cells through regulatory T cells and TGF-β.A common atopy-associated variant in the Th2 cytokine locus control region impacts transcriptional regulation and alters SMAD3 and SP1 binding
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Smad3 -signalling and Th2 cytokines in normal mouse airways and in a mouse model of asthma.
description
2007 nî lūn-bûn
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2007年の論文
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2007年学术文章
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2007年学术文章
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name
Smad3 -signalling and Th2 cyto ...... nd in a mouse model of asthma.
@en
type
label
Smad3 -signalling and Th2 cyto ...... nd in a mouse model of asthma.
@en
prefLabel
Smad3 -signalling and Th2 cyto ...... nd in a mouse model of asthma.
@en
P2093
P2860
P356
P1476
Smad3 -signalling and Th2 cyto ...... nd in a mouse model of asthma.
@en
P2093
Antti I Lauerma
Guoying Wang
Harri T Alenius
Henrik J Wolff
Marina S Leino
Minna Anthoni
P2860
P304
P356
10.7150/IJBS.3.477
P407
P577
2007-11-24T00:00:00Z